Evidence map›Paper›PMID 37298421›Full record

ReviewInternational journal of molecular sciences2023

The Aging Enteric Nervous System.

Tinh Thi Nguyen, Peter Baumann, Oliver Tüscher, Sandra Schick, Kristina Endres

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 31 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
31citing papers in PubMed, 1 pooled it
10.3field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

31 citing papers in PubMed, 1 synthesis or guideline pooled it, 38 citations in OpenAlex.

  1. Pooled it
  2. Review
  3. Article
  4. Article
  5. Review
  6. Review
  7. Review
  8. Review
  9. Review
  10. Age-Related Transcriptomic Changes in the Vermiform Appendix.International journal of molecular sciences · 2025
    Article
  11. Review
  12. Article
  13. Review
  14. Review
  15. Review
  16. Article
  17. Review
  18. Review
  19. Review
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Tinh Thi NguyenDepartment of Psychiatry and Psychotherapy, University Medical Center Mainz, 55131 Mainz, Germany.ORCID 0000-0003-4676-3225
Peter BaumannChromosome Dynamics, Telomeres & Aging Group, Faculty of Biology and Institute of Molecular Biology, 55128 Mainz, Germany.
Oliver TüscherDepartment of Psychiatry and Psychotherapy, University Medical Center Mainz, 55131 Mainz, Germany.
Sandra SchickChromatin Regulation Group, Institute of Molecular Biology, 55128 Mainz, Germany.ORCID 0000-0002-4061-2381
Kristina EndresDepartment of Psychiatry and Psychotherapy, University Medical Center Mainz, 55131 Mainz, Germany.ORCID 0000-0002-1099-8287
Johannes Gutenberg University Mainz · DEInstitute of Molecular Biology · DE

Funding

The Ministry of Science and Health (MWG) of Rhineland-Palatinate, Germany xx
6 · The paper itself

Abstract

The gut and the brain communicate via the nervous system, hormones, microbiota-mediated substances, and the immune system. These intricate interactions have led to the term "gut-brain axis". Unlike the brain-which is somewhat protected-the gut is exposed to a variety of factors throughout life and, consequently, might be either more vulnerable or better adapted to respond to these challenges. Alterations in gut function are common in the elder population and associated with many human pathologies, including neurodegenerative diseases. Different studies suggest that changes in the nervous system of the gut, the enteric nervous system (ENS), during aging may result in gastrointestinal dysfunction and initiate human pathologies of the brain via its interconnection with the gut. This review aims at summarizing the contribution of normal cellular aging to the age-associated physiological changes of the ENS. Morphological alterations and degeneration of the aging ENS are observed in different animal models and humans, albeit with considerable variability. The aging phenotypes and pathophysiological mechanisms of the aging ENS have highlighted the involvement of enteric neurons in age-related diseases of the central nervous system such as Alzheimer's or Parkinson's disease. To further elucidate such mechanisms, the ENS constitutes a promising source of material for diagnosis and therapeutic predictions, as it is more accessible than the brain.

Indexed as

Enteric Nervous SystemParkinson DiseaseAgedAgingAnimalsBrainCentral Nervous SystemHumansagingenteric nervous systemgutgut–brain axismicrobiotaneurodegenerative disease

Identifiers

PMID37298421
PMCPMC10253713
OpenAlexW4380225007

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.