Evidence mapPaperPMID 37369508Full record

ArticleIn vivo (Athens, Greece)

The EGF Motif With CXDXXXXYXCXC Sequence Suppresses Fibrosis in a Mouse Skin Wound Model.

Hisataka Kitano, Tomomi Ishikawa, Yoh Masaoka, Kazuhiro Komiyama, Mamiko Takahashi, Chiaki Hidai

Open access · diamondAbstract read
In one paragraph

Article in In vivo (Athens, Greece). The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
field-weighted citation impact, top 89% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 0 citations in OpenAlex.

  1. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Hisataka Kitano *Division of Oral Surgery, Nihon University School of Medicine, Tokyo, Japan.
Tomomi Ishikawa *Division of Oral Surgery, Nihon University School of Medicine, Tokyo, Japan.
Yoh MasaokaDivision of Physiology, Nihon University School of Medicine, Tokyo, Japan.
Kazuhiro KomiyamaDivision of Physiology, Nihon University School of Medicine, Tokyo, Japan.
Mamiko TakahashiDivision of Physiology, Nihon University School of Medicine, Tokyo, Japan.
Chiaki HidaiDivision of Medical Education, Nihon University School of Medicine, Tokyo, Japan hidai.chiaki@nihon-u.ac.jp.
Nihon University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

BACKGROUND/

aimFibrosis is an essential process for wound healing, but excessive fibrosis, such as keloids and hypertrophic scars, can cause cosmetic and functional problems. These lesions are caused by abnormal deposition and shrinkage of collagen fibers. The light chain of FIX, a plasma protein essential for hemostasis, has the amino acid sequence CXDXXXXYXCXC in the EGF domain. Peptides containing this sequence inhibited stromal growth in a mouse transplant tumor model. In this study, the effect of the FIX light chain on wound healing was studied. MATERIALS AND

methodsA full-layer wound was made on the back of each mouse, and cDNA encoding the light chain of mouse FIX (F9-LC) in an expression vector was injected locally once each week using a non-viral vector. Histochemical analysis of the wound was then performed to assess the effects on wound healing. Moreover, the effect of F9-LC on fibroblasts was studied in vitro.

resultsMacroscopic observation showed that wounds with forced expression of F9-LC appeared flatter and had fewer wrinkles than control wounds. Tissue collagen staining and immunostaining revealed that administration of F9-LC suppressed collagen 1 and 3 deposition and decreased α-smooth muscle actin expression. Electron microscopy revealed sparse and disorganized collagen fibers in the F9-LC-treated mice. In experiments using fibroblasts, addition of a recombinant protein of the FIX light chain disrupted the typical spindle shape and alignment of fibroblasts.

conclusionF9-LC is a new candidate for use in treatments to regulate excessive fibrosis and contraction in wound healing.

Indexed as

Epidermal Growth FactorSoft Tissue InjuriesAnimalsCollagenDisease Models, AnimalFibroblastsFibrosisMiceSkinWound HealingCollagenEpidermal Growth Factorcoagulation factor IXCXDXXXXYXCXCdevelopmental endothelial locus 1EGF motiffibrosis

Identifiers

PMID37369508
PMCPMC10347959
OpenAlexW4382345002

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.