Evidence map›Paper›PMID 37389261›Full record

ArticleChemical science2023

Activation of the cGAS-STING pathway by a mitochondrial DNA-targeted emissive rhodium(iii) metallointercalator.

Yue Zheng, Xiao-Xiao Chen, Dong-Yang Zhang, Wen-Jin Wang, Kun Peng, Zhi-Yuan Li, Zong-Wan Mao, Cai-Ping Tan

Abstract read
In one paragraph

Article in Chemical science, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed.

  1. Review
  2. Review
  3. Article
  4. Article
  5. Review
  6. Review
  7. Article
  8. Article
  9. Agonists and Inhibitors of the cGAS-STING Pathway.Molecules (Basel, Switzerland) · 2024
    Review
  10. Review
  11. Mitochondrial DNA and Inflammation in Alzheimer's Disease.Current issues in molecular biology · 2023
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Yue ZhengMOE Key Laboratory of Bioinorganic and Synthetic Chemistry, School of Chemistry, State Key Laboratory of Oncology in South China, Sun Yat-Sen University Guangzhou 510006 P. R. China cesmzw@mail.sysu.edu.cn tancaip@mail.sysu.edu.cn.ORCID https://orcid.org/0000-0003-4720-7378
Xiao-Xiao ChenMOE Key Laboratory of Bioinorganic and Synthetic Chemistry, School of Chemistry, State Key Laboratory of Oncology in South China, Sun Yat-Sen University Guangzhou 510006 P. R. China cesmzw@mail.sysu.edu.cn tancaip@mail.sysu.edu.cn.
Dong-Yang ZhangMOE Key Laboratory of Bioinorganic and Synthetic Chemistry, School of Chemistry, State Key Laboratory of Oncology in South China, Sun Yat-Sen University Guangzhou 510006 P. R. China cesmzw@mail.sysu.edu.cn tancaip@mail.sysu.edu.cn.
Wen-Jin WangMOE Key Laboratory of Bioinorganic and Synthetic Chemistry, School of Chemistry, State Key Laboratory of Oncology in South China, Sun Yat-Sen University Guangzhou 510006 P. R. China cesmzw@mail.sysu.edu.cn tancaip@mail.sysu.edu.cn.
Kun PengMOE Key Laboratory of Bioinorganic and Synthetic Chemistry, School of Chemistry, State Key Laboratory of Oncology in South China, Sun Yat-Sen University Guangzhou 510006 P. R. China cesmzw@mail.sysu.edu.cn tancaip@mail.sysu.edu.cn.ORCID https://orcid.org/0000-0001-6885-9899
Zhi-Yuan LiMOE Key Laboratory of Bioinorganic and Synthetic Chemistry, School of Chemistry, State Key Laboratory of Oncology in South China, Sun Yat-Sen University Guangzhou 510006 P. R. China cesmzw@mail.sysu.edu.cn tancaip@mail.sysu.edu.cn.
Zong-Wan MaoMOE Key Laboratory of Bioinorganic and Synthetic Chemistry, School of Chemistry, State Key Laboratory of Oncology in South China, Sun Yat-Sen University Guangzhou 510006 P. R. China cesmzw@mail.sysu.edu.cn tancaip@mail.sysu.edu.cn.ORCID https://orcid.org/0000-0001-7131-1154
Cai-Ping TanMOE Key Laboratory of Bioinorganic and Synthetic Chemistry, School of Chemistry, State Key Laboratory of Oncology in South China, Sun Yat-Sen University Guangzhou 510006 P. R. China cesmzw@mail.sysu.edu.cn tancaip@mail.sysu.edu.cn.ORCID https://orcid.org/0000-0002-7652-6645

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The cyclic GMP-AMP synthase (cGAS)-stimulator of interferon (STING) pathway is a key mediator of innate immunity involved in cancer development and treatment. The roles of mitochondrial DNA (mtDNA) in cancer immunotherapy have gradually emerged. Herein, we report a highly emissive rhodium(iii) complex (Rh-Mito) as the mtDNA intercalator. Rh-Mito can specifically bind to mtDNA to cause the cytoplasmic release of mtDNA fragments to activate the cGAS-STING pathway. Moreover, Rh-Mito activates the mitochondrial retrograde signaling by disturbing the key metabolites involved in epigenetic modifications, which alters the nuclear genome methylation landscape to influence the expression of genes related to immune signaling pathways. Finally, we demonstrate that ferritin-encapsulated Rh-Mito elicits potent anticancer activities and evokes intense immune responses

Identifiers

PMID37389261
PMCPMC10306090

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.