Evidence mapPaperPMID 37405259Full record

ReviewFrontiers in molecular biosciences2023

Psoriasis immunometabolism: progress on metabolic biomarkers and targeted therapy.

Evangelia Sarandi, Sabine Krueger-Krasagakis, Dimitris Tsoukalas, Polytimi Sidiropoulou, George Evangelou, Maria Sifaki, Gottfried Rudofsky, Nikolaos Drakoulis, Aristidis Tsatsakis

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in molecular biosciences, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 27 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
27citing papers in PubMed, 1 pooled it
5.8field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

27 citing papers in PubMed, 1 synthesis or guideline pooled it, 35 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 6 institutions in 3 countries.

Evangelia SarandiLaboratory of Toxicology and Forensic Sciences, Medical School, University of Crete, Heraklion, Greece.
Sabine Krueger-KrasagakisDermatology Department, University Hospital of Heraklion, Heraklion, Greece.
Dimitris TsoukalasMetabolomic Medicine, Health Clinics for Autoimmune and Chronic Diseases, Athens, Greece.
Polytimi Sidiropoulou1st Department of Dermatology-Venereology, Faculty of Medicine, "A. Sygros" Hospital, National and Kapodistrian University of Athens, Athens, Greece.
George EvangelouDermatology Department, University Hospital of Heraklion, Heraklion, Greece.
Maria SifakiResearch Group of Clinical Pharmacology and Pharmacogenomics, Faculty of Pharmacy, School of Health Sciences, National and Kapodistrian University of Athens, Athens, Greece.
Gottfried RudofskyClinic of Endocrinology and Metabolic Disorders, Cantonal Hospital Olten, Olten, Switzerland.
Nikolaos DrakoulisResearch Group of Clinical Pharmacology and Pharmacogenomics, Faculty of Pharmacy, School of Health Sciences, National and Kapodistrian University of Athens, Athens, Greece.
Aristidis TsatsakisLaboratory of Toxicology and Forensic Sciences, Medical School, University of Crete, Heraklion, Greece.
National and Kapodistrian University of Athens · GRUniversity Hospital of Heraklion · GRUniversity of Crete · GRAndreas Sygros Hospital · GREuropean School of Molecular Medicine · ITKantonsschule Olten · CH

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Psoriasis is a common inflammatory disease that affects mainly the skin. However, the moderate to severe forms have been associated with several comorbidities, such as psoriatic arthritis, Crohn's disease, metabolic syndrome and cardiovascular disease. Keratinocytes and T helper cells are the dominant cell types involved in psoriasis development via a complex crosstalk between epithelial cells, peripheral immune cells and immune cells residing in the skin. Immunometabolism has emerged as a potent mechanism elucidating the aetiopathogenesis of psoriasis, offering novel specific targets to diagnose and treat psoriasis early. The present article discusses the metabolic reprogramming of activated T cells, tissue-resident memory T cells and keratinocytes in psoriatic skin, presenting associated metabolic biomarkers and therapeutic targets. In psoriatic phenotype, keratinocytes and activated T cells are glycolysis dependent and are characterized by disruptions in the TCA cycle, the amino acid metabolism and the fatty acid metabolism. Upregulation of the mammalian target of rapamycin (mTOR) results in hyperproliferation and cytokine secretion by immune cells and keratinocytes. Metabolic reprogramming through the inhibition of affected metabolic pathways and the dietary restoration of metabolic imbalances may thus present a potent therapeutic opportunity to achieve long-term management of psoriasis and improved quality of life with minimum adverse effects.

Indexed as

biomarkersglycolysiskeratinocytelipid metabolismmetabolic targetspsoriasisTCAT cell

Identifiers

PMID37405259
PMCPMC10317015
OpenAlexW4381189849

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.