Evidence mapPaperPMID 37445845Full record

ReviewInternational journal of molecular sciences2023

Unlocking the Potential of Arginine Deprivation Therapy: Recent Breakthroughs and Promising Future for Cancer Treatment.

Yu-De Chu, Ming-Wei Lai, Chau-Ting Yeh

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed
8.8field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 33 citations in OpenAlex.

  1. Review
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  11. Extract ofToxins · 2025
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 3 institutions in 1 country.

Yu-De ChuLiver Research Center, Chang Gung Memorial Hospital, Linkou Branch, Taoyuan 333, Taiwan.
Ming-Wei LaiLiver Research Center, Chang Gung Memorial Hospital, Linkou Branch, Taoyuan 333, Taiwan.
Chau-Ting YehLiver Research Center, Chang Gung Memorial Hospital, Linkou Branch, Taoyuan 333, Taiwan.
Chang Gung Memorial Hospital · TWChang Gung University · TWLinkou Chang Gung Memorial Hospital · TW

Funding

Linkou Chang Gung Memorial Hospital CMRPG3M0891
6 · The paper itself

Abstract

Arginine is a semi-essential amino acid that supports protein synthesis to maintain cellular functions. Recent studies suggest that arginine also promotes wound healing, cell division, ammonia metabolism, immune system regulation, and hormone biosynthesis-all of which are critical for tumor growth. These discoveries, coupled with the understanding of cancer cell metabolic reprogramming, have led to renewed interest in arginine deprivation as a new anticancer therapy. Several arginine deprivation strategies have been developed and entered clinical trials. The main principle behind these therapies is that arginine auxotrophic tumors rely on external arginine sources for growth because they carry reduced key arginine-synthesizing enzymes such as argininosuccinate synthase 1 (ASS1) in the intracellular arginine cycle. To obtain anticancer effects, modified arginine-degrading enzymes, such as PEGylated recombinant human arginase 1 (rhArg1-PEG) and arginine deiminase (ADI-PEG 20), have been developed and shown to be safe and effective in clinical trials. They have been tried as a monotherapy or in combination with other existing therapies. This review discusses recent advances in arginine deprivation therapy, including the molecular basis of extracellular arginine degradation leading to tumor cell death, and how this approach could be a valuable addition to the current anticancer arsenal.

Indexed as

ArginineNeoplasmsArgininosuccinate SynthaseCell DeathCell Line, TumorHumansHydrolasesPolyethylene GlycolsArginineArgininosuccinate SynthaseHydrolasesPolyethylene GlycolsADI-PEG 20arginine auxotrophic cancerarginine deprivation cancer therapyBCT-100biomarkercirculating argininerhArg1-PEG

Identifiers

PMID37445845
PMCPMC10341449
OpenAlexW4382458948

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.