Evidence map›Paper›PMID 37511575›Full record

ArticleInternational journal of molecular sciences2023

Assessing the Link between Diabetic Metabolic Dysregulation and Breast Cancer Progression.

Samrein B M Ahmed, Nada Radwan, Sara Amer, Narjes Saheb Sharif-Askari, Amena Mahdami, Kamel A Samara, Rabih Halwani, Herbert F Jelinek

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
1.4field-weighted citation impact, top 19% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 9 citations in OpenAlex.

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  7. The influence of diabetes mellitus on blood vessels' amounts and immune status in case of breast cancer.Romanian journal of morphology and embryology = Revue roumaine de morphologie et embryologie
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 2 institutions in 2 countries.

Samrein B M AhmedResearch Institute of Medical and Health Sciences, University of Sharjah, Sharjah 27272, United Arab Emirates.
Nada RadwanResearch Institute of Medical and Health Sciences, University of Sharjah, Sharjah 27272, United Arab Emirates.ORCID 0000-0001-6877-6911
Sara AmerResearch Institute of Medical and Health Sciences, University of Sharjah, Sharjah 27272, United Arab Emirates.
Narjes Saheb Sharif-AskariResearch Institute of Medical and Health Sciences, University of Sharjah, Sharjah 27272, United Arab Emirates.ORCID 0000-0003-0482-6777
Amena MahdamiResearch Institute of Medical and Health Sciences, University of Sharjah, Sharjah 27272, United Arab Emirates.
Kamel A SamaraCollege of Medicine, University of Sharjah, Sharjah 27272, United Arab Emirates.ORCID 0000-0002-8536-6495
Rabih HalwaniResearch Institute of Medical and Health Sciences, University of Sharjah, Sharjah 27272, United Arab Emirates.
Herbert F JelinekDepartment of Biomedical Engineering and Health Engineering Innovation Center, Khalifa University, Abu Dhabi 127788, United Arab Emirates.
University of Sharjah · AEKhalifa University of Science and Technology · AE

Funding

The present study was supported by grants from the University of Sharjah (grants no. 16010901011 and 1801090143), UAE 16010901011 and 1801090143
6 · The paper itself

Abstract

Diabetes mellitus is a burdensome disease that affects various cellular functions through altered glucose metabolism. Several reports have linked diabetes to cancer development; however, the exact molecular mechanism of how diabetes-related traits contribute to cancer progression is not fully understood. The current study aimed to explore the molecular mechanism underlying the potential effect of hyperglycemia combined with hyperinsulinemia on the progression of breast cancer cells. To this end, gene dysregulation induced by the exposure of MCF7 breast cancer cells to hyperglycemia (HG), or a combination of hyperglycemia and hyperinsulinemia (HGI), was analyzed using a microarray gene expression assay. Hyperglycemia combined with hyperinsulinemia induced differential expression of 45 genes (greater than or equal to two-fold), which were not shared by other treatments. On the other hand, in silico analysis performed using a publicly available dataset (GEO: GSE150586) revealed differential upregulation of 15 genes in the breast tumor tissues of diabetic patients with breast cancer when compared with breast cancer patients with no diabetes.

Indexed as

Breast NeoplasmsDiabetes MellitusDiabetes Mellitus, Type 2HyperglycemiaHyperinsulinismFemaleGlycemic IndexHumansbreast cancerdiabetesgene dysregulationhyperglycemiahyperinsulinemia

Identifiers

PMID37511575
PMCPMC10380477
OpenAlexW4385161840

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.