Evidence mapPaperPMID 37528345Full record

ArticleBMC cancer2023

Salidroside regulates tumor microenvironment of non-small cell lung cancer via Hsp70/Stub1/Foxp3 pathway in Tregs.

Zexin Wen, Tong Liu, Yanli Zhang, Qiujuan Yue, Hang Meng, Yijie He, Yi Yang, Minghao Li, Jianwen Zheng, Wei Lin

Open access · goldAbstract read
In one paragraph

Article in BMC cancer, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed
3.2field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 21 citations in OpenAlex.

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  18. Mechanism of salidroside in tumor suppression through the miRNA-mRNA signaling axis.Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences · 2024
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 3 institutions in 1 country.

Zexin Wen *Department of Medicine, Xizang Minzu University, Xianyang, Shaanxi, China.
Tong Liu *Department of Medicine, Xizang Minzu University, Xianyang, Shaanxi, China.
Yanli ZhangDepartment of Medicine, Xizang Minzu University, Xianyang, Shaanxi, China.
Qiujuan YueDepartment of Medicine, Xizang Minzu University, Xianyang, Shaanxi, China.
Hang MengDepartment of Medicine, Xizang Minzu University, Xianyang, Shaanxi, China.
Yijie HeDepartment of Medicine, Xizang Minzu University, Xianyang, Shaanxi, China.
Yi YangDepartment of Medicine, Xizang Minzu University, Xianyang, Shaanxi, China.
Minghao LiDepartment of Oncology, The First Affiliated Hospital of Shandong First Medical University, Shandong Provincial Qianfoshan Hospital, Jinan, China.
Jianwen ZhengDepartment of Medicine, Xizang Minzu University, Xianyang, Shaanxi, China. 1024559902@qq.com.
Wei LinDepartment of Oncology, The First Affiliated Hospital of Shandong First Medical University, Shandong Provincial Qianfoshan Hospital, Jinan, China. linw1978@163.com.
Xizang Minzu University · CNShandong First Medical University · CNShandong Provincial Hospital · CN

Funding

the National Natural Science Foundation of China 81860719
6 · The paper itself

Abstract

backgroundThe treatment of non-small cell lung cancer (NSCLC) is challenging due to immune tolerance and evasion. Salidroside (SAL) is an extract in traditional Chinese medicine and has a potential antitumor effect. However, the mechanism of SAL in regulating the immunological microenvironment of NSCLC is yet to be clarified.

methodsThe mouse model with Lewis lung cancer cell line (3LL) in C57BL/6 mice was established. And then, the percentage of tumor-infiltrating T cell subsets including Treg was detected in tumor-bearing mice with or without SAL treatment. In vitro, the effect of SAL on the expression of IL-10, Foxp3 and Stub1 and the function of Treg were detected by flow cytometry. Network pharmacology prediction and molecular docking software were used to predict the target of SAL and intermolecular interaction. Furthermore, the effect of SAL on the expression of Hsp70 and the co-localization of Stub1-Foxp3 in Treg was confirmed by flow cytometry and confocal laser microscopy. Finally, Hsp70 inhibitor was used to verify the above molecular expression.

resultsWe discovered that SAL treatment inhibits the growth of tumor cells by decreasing the percentage of tumor-infiltrated CD4

conclusionsSAL inhibits tumor growth by regulating the Hsp70/stub1/Foxp3 pathway in Treg to suppress the function of Treg. It is a new mechanism of SAL for antitumor therapy.

Indexed as

Carcinoma, Non-Small-Cell LungLung NeoplasmsAnimalsForkhead Transcription FactorsGlucosidesMiceMice, Inbred C57BLMolecular Docking SimulationPhenolsT-Lymphocytes, RegulatoryTumor MicroenvironmentUbiquitin-Protein LigasesForkhead Transcription FactorsFoxp3 protein, mouseGlucosidesPhenolsrhodiolosideUbiquitin-Protein LigasesNon-small cell lung cancerSalidrosideStub1TregTumor microenvironment

Identifiers

PMID37528345
PMCPMC10391887
OpenAlexW4385458602

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.