Evidence mapPaperPMID 37538867Full record

ArticleChinese herbal medicines2023

Mingning Ding, Fei Zhou, Yijie Li, Chuanyang Liu, Yiqing Gu, Jianzhi Wu, Guifang Fan, Yajing Li, Xiaojiaoyang Li

Open access · goldAbstract read
In one paragraph

Article in Chinese herbal medicines, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed
5.4field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

19 citing papers in PubMed, 25 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Unveiling bioactive components inChinese herbal medicines · 2025
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  6. Schottky barrier in pea-like Au@BiMaterials today. Bio · 2025
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  7. Article
  8. Anthraquinones ofChinese herbal medicines · 2025
    Article
  9. Review
  10. NSP6 of SARS-CoV-2 Dually Regulates Autophagic-Lysosomal Degradation.International journal of molecular sciences · 2025
    Article
  11. Article
  12. Article
  13. Frontiers in pharmacology · 2025
    Article
  14. Article
  15. Review
  16. Article
  17. Article
  18. Article
  19. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 1 institution in 1 country.

Mingning DingSchool of Life Sciences, Beijing University of Chinese Medicine, Beijing 100029, China.
Fei ZhouSchool of Life Sciences, Beijing University of Chinese Medicine, Beijing 100029, China.
Yijie LiSchool of Life Sciences, Beijing University of Chinese Medicine, Beijing 100029, China.
Chuanyang LiuSchool of Traditional Chinese Medicine, Beijing University of Chinese Medicine, Beijing 100029, China.
Yiqing GuSchool of Chinese Materia Medica, Beijing University of Chinese Medicine, Beijing 100029, China.
Jianzhi WuSchool of Life Sciences, Beijing University of Chinese Medicine, Beijing 100029, China.
Guifang FanSchool of Chinese Materia Medica, Beijing University of Chinese Medicine, Beijing 100029, China.
Yajing LiSchool of Life Sciences, Beijing University of Chinese Medicine, Beijing 100029, China.
Xiaojiaoyang LiSchool of Life Sciences, Beijing University of Chinese Medicine, Beijing 100029, China.
Beijing University of Chinese Medicine · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Objective: Methods: High-performance liquid chromatography (HPLC) was used to identify the major components of CS water extract. Mice were fed with a high-fat and sugar-water (HFSW) diet to induce hepatic steatosis and then treated with CS. The anti-NAFLD effect was determined by measuring serum biomarkers and histopathology staining. Additionally, the effects of CS on cell viability and lipid metabolism in oleic acid and palmitic acid (OAPA)-treated HepG2 cells were measured. The expression of essential genes and proteins involved in lipid metabolism and autophagy signalings were measured to uncover the underlying mechanism. Results: Five compounds, including aurantio-obtusin, rubrofusarin gentiobioside, cassiaside C, emodin and rhein were simultaneously identified in CS extract. CS not only improved the diet-induced hepatic steatosis Conclusion: Our study suggested that CS effectively protected liver steatosis via decreasing FASN-related fatty acid synthesis and activating AMPK-mediated autophagy, which might become a promising therapeutic strategy for relieving NAFLD.

Indexed as

AMPKautophagyCassiae SemenCassia obtusifolia L.Cassia tora L.fatty acid synthesisnon-alcoholic fatty acid disease

Identifiers

PMID37538867
PMCPMC10394324
OpenAlexW4353047550

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.