Evidence map›Paper›PMID 37545519›Full record

ArticleFrontiers in immunology2023

Exome sequencing to explore the possibility of predicting genetic susceptibility to the joint occurrence of polycystic ovary syndrome and Hashimoto's thyroiditis.

Natalia Zeber-Lubecka, Katarzyna Suchta, Maria Kulecka, Anna Kluska, Magdalena Piątkowska, Michal J Dabrowski, Katarzyna Jankowska, Monika Grymowicz, Roman Smolarczyk, Ewa E Hennig

Abstract read
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Article in Frontiers in immunology, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed.

  1. Heterozygous pathogenic variants inOpen life sciences · 2026
    Article
  2. Review
  3. Article
  4. Review
  5. Polycystic Ovary Syndrome and Oxidative Stress-From Bench to Bedside.International journal of molecular sciences · 2023
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Natalia Zeber-LubeckaDepartment of Gastroenterology, Hepatology and Clinical Oncology, Centre of Postgraduate Medical Education, Warsaw, Poland.
Katarzyna SuchtaDepartment of Gynaecological Endocrinology, Medical University of Warsaw, Warsaw, Poland.
Maria KuleckaDepartment of Gastroenterology, Hepatology and Clinical Oncology, Centre of Postgraduate Medical Education, Warsaw, Poland.
Anna KluskaDepartment of Genetics, Maria Sklodowska-Curie National Research Institute of Oncology, Warsaw, Poland.
Magdalena PiątkowskaDepartment of Genetics, Maria Sklodowska-Curie National Research Institute of Oncology, Warsaw, Poland.
Michal J DabrowskiInstitute of Computer Science, Polish Academy of Sciences, Warsaw, Poland.
Katarzyna JankowskaDepartment of Endocrinology, Centre of Postgraduate Medical Education, Warsaw, Poland.
Monika GrymowiczDepartment of Gynaecological Endocrinology, Medical University of Warsaw, Warsaw, Poland.
Roman SmolarczykDepartment of Gynaecological Endocrinology, Medical University of Warsaw, Warsaw, Poland.
Ewa E HennigDepartment of Gastroenterology, Hepatology and Clinical Oncology, Centre of Postgraduate Medical Education, Warsaw, Poland.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

A large body of evidence indicates that women with polycystic ovary syndrome (PCOS) have a higher risk of developing Hashimoto's thyroiditis (HT) than healthy individuals. Given the strong genetic impact on both diseases, common predisposing genetic factors are possibly involved but are not fully understood. Here, we performed whole-exome sequencing (WES) for 250 women with sporadic PCOS, HT, combined PCOS and HT (PCOS+HT), and healthy controls to explore the genetic background of the joint occurrence of PCOS and HT. Based on relevant comparative analyses, multivariate logistic regression prediction modeling, and the most informative feature selection using the Monte Carlo feature selection and interdependency discovery algorithm, 77 variants were selected for further validation by TaqMan genotyping in a group of 533 patients. In the allele frequency test, variants in

Indexed as

Hashimoto DiseasePolycystic Ovary SyndromeApoptosis Regulatory ProteinsExome SequencingFemaleGene FrequencyGenetic Predisposition to DiseaseHumansRepressor ProteinsApoptosis Regulatory ProteinsHIF3A protein, humanRepressor Proteinsautoimmune thyroid diseaseciliafemale meiosisHashimoto thyroiditis (HT)immune responsepolycystic ovary syndromeprediction modelwhole-exome sequencing (WES)

Identifiers

PMID37545519
PMCPMC10397507

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.