Evidence map›Paper›PMID 37686720›Full record

ReviewNutrients2023

Modulation of microRNAs through Lifestyle Changes in Alzheimer's Disease.

Paola Pinto-Hernandez, Juan Castilla-Silgado, Almudena Coto-Vilcapoma, Manuel Fernández-Sanjurjo, Benjamín Fernández-García, Cristina Tomás-Zapico, Eduardo Iglesias-Gutiérrez

Open access · goldAbstract readReview
In one paragraph

Review in Nutrients, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed, 1 pooled it
4.1field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 1 synthesis or guideline pooled it, 17 citations in OpenAlex.

  1. Pooled it
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Paola Pinto-HernandezDepartment of Functional Biology, Physiology, University of Oviedo, 33006 Asturias, Spain.
Juan Castilla-SilgadoDepartment of Functional Biology, Physiology, University of Oviedo, 33006 Asturias, Spain.ORCID 0000-0002-6266-8247
Almudena Coto-VilcapomaDepartment of Functional Biology, Physiology, University of Oviedo, 33006 Asturias, Spain.ORCID 0000-0002-9856-6939
Manuel Fernández-SanjurjoDepartment of Functional Biology, Physiology, University of Oviedo, 33006 Asturias, Spain.ORCID 0000-0003-4687-3379
Benjamín Fernández-GarcíaHealth Research Institute of the Principality of Asturias (ISPA), 33011 Asturias, Spain.ORCID 0000-0002-2391-680X
Cristina Tomás-ZapicoDepartment of Functional Biology, Physiology, University of Oviedo, 33006 Asturias, Spain.ORCID 0000-0001-7823-0410
Eduardo Iglesias-GutiérrezDepartment of Functional Biology, Physiology, University of Oviedo, 33006 Asturias, Spain.ORCID 0000-0002-3063-1562
Universidad de Oviedo · ES

Funding

Foundation for the Promotion of Applied Scientific Research and Technology in Asturias AYUD/2021/5134Foundation for the Promotion of Applied Scientific Research and Technology in Asturias AYUD/2021/57540
6 · The paper itself

Abstract

Lifestyle factors, including diet and physical activity (PA), are known beneficial strategies to prevent and delay Alzheimer's disease (AD) development. Recently, microRNAs have emerged as potential biomarkers in multiple diseases, including AD. The aim of this review was to analyze the available information on the modulatory effect of lifestyle on microRNA expression in AD. Few studies have addressed this question, leaving important gaps and limitations: (1) in human studies, only circulating microRNAs were analyzed; (2) in mice studies, microRNA expression was only analyzed in brain tissue; (3) a limited number of microRNAs was analyzed; (4) no human nutritional intervention studies were conducted; and (5) PA interventions in humans and mice were poorly detailed and only included aerobic training. Despite this, some conclusions could be drawn. Circulating levels of let-7g-5p, miR-107, and miR-144-3p were associated with overall diet quality in mild cognitive impairment patients. In silico analysis showed that these microRNAs are implicated in synapse formation, microglia activation, amyloid beta accumulation, and pro-inflammatory pathways, the latter also being targeted by miR-129-5p and miR-192-5p, whose circulating levels are modified by PA in AD patients. PA also modifies miR-132, miR-15b-5p, miR-148b-3p, and miR-130a-5p expression in mice brains, which targets are related to the regulation of neuronal activity, ageing, and pro-inflammatory pathways. This supports the need to further explore lifestyle-related miRNA changes in AD, both as biomarkers and therapeutic targets.

Indexed as

Alzheimer DiseaseCirculating MicroRNAMicroRNAsAmyloid beta-PeptidesAnimalsHumansLife StyleMiceAmyloid beta-PeptidesCirculating MicroRNAMicroRNAsMirn129 microRNA, humancognitive impairmentdietepigenetic regulationlifestyle interventionsphysical exercise

Identifiers

PMID37686720
PMCPMC10490103
OpenAlexW4386098400

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.