Evidence map›Paper›PMID 37727490›Full record

ReviewWorld journal of clinical cases2023

Mechanism and recent updates on insulin-related disorders.

Shashank Kumar, Sabyasachi Senapati, Neetu Bhattacharya, Amit Bhattacharya, Shashank Kumar Maurya, Hadiya Husain, Jasvinder Singh Bhatti, Abhay Kumar Pandey

Abstract readReview
In one paragraph

Review in World journal of clinical cases, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Article
  3. Review
  4. Chemokine CX3CL1 (Fractalkine) Signaling and Diabetic Encephalopathy.International journal of molecular sciences · 2024
    Review
  5. Article
  6. Review
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Shashank KumarDepartment of Biochemistry, Central University of Punjab, Bathinda 151401, Punjab, India.
Sabyasachi SenapatiDepartment of Human Genetics and Molecular Medicine, Central University of Punjab, Bathinda 151401, Punjab, India.
Neetu BhattacharyaDepartment of Zoology, Dyal Singh College, University of Delhi, New Delhi 110003, India.
Amit BhattacharyaDepartment of Zoology, Ramjas College, University of Delhi, New Delhi 110007, India.
Shashank Kumar MauryaDepartment of Zoology, University of Delhi, New Delhi 110007, India.
Hadiya HusainDepartment of Zoology, University of Lucknow, Lucknow 226007, India.
Jasvinder Singh BhattiDepartment of Human Genetics and Molecular Medicine, Central University of Punjab, Bathinda 151401, Punjab, India.
Abhay Kumar PandeyDepartment of Biochemistry, University of Allahabad, Allahabad (Prayagraj) 211002, India. akpandey23@rediffmail.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Insulin, a small protein with 51 amino acids synthesized by pancreatic β-cells, is crucial to sustain glucose homeostasis at biochemical and molecular levels. Numerous metabolic dysfunctions are related to insulin-mediated altered glucose homeostasis. One of the significant pathophysiological conditions linked to the insulin associated disorder is diabetes mellitus (DM) (type 1, type 2, and gestational). Insulin resistance (IR) is one of the major underlying causes of metabolic disorders despite its association with several physiological conditions. Metabolic syndrome (MS) is another pathophysiological condition that is associated with IR, hypertension, and obesity. Further, several other pathophysiological disorders/diseases are associated with the insulin malfunctioning, which include polycystic ovary syndrome, neuronal disorders, and cancer. Insulinomas are an uncommon type of pancreatic β-cell-derived neuroendocrine tumor that makes up 2% of all pancreatic neoplasms. Literature revealed that different biochemical events, molecular signaling pathways, microRNAs, and microbiota act as connecting links between insulin disorder and associated pathophysiology such as DM, insuloma, neurological disorder, MS, and cancer. In this review, we focus on the insulin-related disorders and the underlying mechanisms associated with the pathophysiology.

Indexed as

CancerDiabetesInsulin disorderMetabolic syndromeNeurological disorderObesity

Identifiers

PMID37727490
PMCPMC10506040

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.