Evidence map›Paper›PMID 37732061›Full record

ArticleArchives of medical science : AMS2023

Serum antinuclear autoantibodies are associated with measures of oxidative stress and lifestyle factors: analysis of LIPIDOGRAM2015 and LIPIDOGEN2015 studies.

Paweł Krzemień, Sławomir Kasperczyk, Maciej Banach, Aleksandra Kasperczyk, Michał Dobrakowski, Tomasz Tomasik, Adam Windak, Mirosław Mastej, Alberico Catapano, Kausik K Ray and 11 more

Abstract read
In one paragraph

Article in Archives of medical science : AMS, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed.

  1. Review
  2. Article
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  4. Review
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

21 authors.

Paweł KrzemieńEuroimmun Polska Sp. z o.o., Wroclaw, Poland.
Sławomir KasperczykDepartment of Biochemistry, Faculty of Medical Sciences in Zabrze, Medical University of Silesia in Katowice Poland.
Maciej BanachDepartment of Hypertension, Medical University of Lodz, Lodz, Poland.
Aleksandra KasperczykDepartment of Biochemistry, Faculty of Medical Sciences in Zabrze, Medical University of Silesia in Katowice Poland.
Michał DobrakowskiDepartment of Biochemistry, Faculty of Medical Sciences in Zabrze, Medical University of Silesia in Katowice Poland.
Tomasz TomasikDepartment of Family Medicine, Jagiellonian University Medical College, Krakow, Poland.
Adam WindakDepartment of Family Medicine, Jagiellonian University Medical College, Krakow, Poland.
Mirosław MastejMastej Medical Center, Jaslo, Poland.
Alberico CatapanoDepartment of Pharmacological Sciences, University of Milano and Multimedica IRCCS, Milano, Italy.
Kausik K RayImperial Centre for Cardiovascular Disease Prevention, Department of Primary Care and Public Health, Imperial College, Kensington, London, United Kingdom.
Dimitri P MikhailidisDepartment of Clinical Biochemistry, Royal Free Hospital, University College London, London, United Kingdom.
Peter P TothCiccarone Center for the Prevention of Cardiovascular Disease, Johns Hopkins University School of Medicine, Baltimore MD, Maryland, USA.
George HowardDepartment of Biostatistics, School of Public Health of Alabama at Birmingham, Birmingham AL, USA.
Gregory Y H LipLiverpool Centre for Cardiovascular Science, University of Liverpool and Liverpool Heart and Chest Hospital, Liverpool L14 3PE, United Kingdom.
Maciej TomaszewskiDivision of Cardiovascular Sciences, Faculty of Biology, Medicine and Health, University of Manchester, Manchester, United Kingdom.
Fadi J CharcharSchool of Health and Life Sciences, Federation University Australia, Ballarat VIC 3350, Victoria, Australia.
Naveed SattarInstitute of Cardiovascular and Medical Science, University of Glasgow, Glasgow, United Kingdom.
Bryan WilliamsNIHR University College London Biomedical Research Centre, University College London and University College London Hospitals NHS Foundation Trust, London, United Kingdom.
Thomas M MacDonaldMEMO Research, University of Dundee, Ninewells Hospital and Medical School, Dundee, United Kingdom.
Peter E PensonSchool of Pharmacy and Biomolecular Sciences, Liverpool John Moores University, Liverpool, United Kingdom.
Jacek J JóźwiakDepartment of Family Medicine and Public Health, Faculty of Medicine, University of Opole, Opole, Poland.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Introduction: Oxidative stress is one of many factors suspected to promote antinuclear autoantibody (ANA) formation. Reactive oxygen species can induce changes in the antigenic structure of macromolecules, causing the immune system to treat them as "neo-antigens" and start production of autoantibodies. This study was designed to evaluate the relationship between oxidative stress markers, lifestyle factors and the detection of ANA. Material and methods: We examined measures of oxidative stress indices of free-radical damage to lipids and proteins, such as total oxidant status (TOS), concentration of protein thiol groups (PSH), and malondialdehyde (MDA), activity of superoxide dismutase (SOD) in 1731 serum samples. The parameters of the non-enzymatic antioxidant system, such as total antioxidant status (TAS) and uric acid (UA) concentration, were also measured and the oxidative stress index (OSI-index) was calculated. All samples were tested for the presence of ANA using an indirect immunofluorescence assay (IIFA). Results: The presence of ANA in women was associated with lower physical activity ( Conclusions: The results indicate that local oxidative stress may be associated with increased probability of ANA formation in a sex-specific manner.

Indexed as

antinuclear autoantibodylifestyle diseasesoxidative stressreactive oxygen species

Identifiers

PMID37732061
PMCPMC10507751

What Socratic holds

Textmetadata
LicenceCC BY-NC-SA
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.