Evidence mapPaperPMID 37760075Full record

ReviewAntioxidants (Basel, Switzerland)2023

The Role of Oxidative Stress in Kidney Injury.

Nejc Piko, Sebastjan Bevc, Radovan Hojs, Robert Ekart

Open access · goldAbstract readReview
In one paragraph

Review in Antioxidants (Basel, Switzerland), 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 40 papers.

0numbers the graph read from it
0cells of the map it votes in
40citing papers in PubMed
12.5field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

40 citing papers in PubMed, 61 citations in OpenAlex.

  1. An HRedox biology · 2026
    Article
  2. Review
  3. The endothelium response to kidney injury.Nature reviews. Nephrology · 2026
    Review
  4. Review
  5. Review
  6. Review
  7. Review
  8. Review
  9. Article
  10. Article
  11. Article
  12. Nephroprotective activity of Eysenhardtia polystachya through antioxidant, HNaunyn-Schmiedeberg's archives of pharmacology · 2025
    Article
  13. Article
  14. Article
  15. Review
  16. Article
  17. The death of a myth: Females are not resistant to acute kidney injury.Clinical science (London, England : 1979) · 2025
    Article
  18. Article
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Nejc PikoDepartment of Dialysis, Clinic for Internal Medicine, University Medical Centre, 2000 Maribor, Slovenia.ORCID 0000-0003-1314-4428
Sebastjan BevcDepartment of Nephrology, Clinic for Internal Medicine, University Medical Centre, 2000 Maribor, Slovenia.ORCID 0000-0003-2313-1559
Radovan HojsDepartment of Nephrology, Clinic for Internal Medicine, University Medical Centre, 2000 Maribor, Slovenia.
Robert EkartDepartment of Dialysis, Clinic for Internal Medicine, University Medical Centre, 2000 Maribor, Slovenia.ORCID 0000-0002-7395-752X
University of Maribor · SIUniversity Clinical Centre Maribor · SI

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Acute kidney injury and chronic kidney disease are among the most common non-communicable diseases in the developed world, with increasing prevalence. Patients with acute kidney injury are at an increased risk of developing chronic kidney disease. One of kidney injury's most common clinical sequelae is increased cardiovascular morbidity and mortality. In recent years, new insights into the pathophysiology of renal damage have been made. Oxidative stress is the imbalance favoring the increased generation of ROS and/or reduced body's innate antioxidant defense mechanisms and is of pivotal importance, not only in the development and progression of kidney disease but also in understanding the enhanced cardiovascular risk in these patients. This article summarizes and emphasizes the role of oxidative stress in acute kidney injury, various forms of chronic kidney disease, and also in patients on renal replacement therapy (hemodialysis, peritoneal dialysis, and after kidney transplant). Additionally, the role of oxidative stress in the development of drug-related nephrotoxicity and also in the development after exposure to various environmental and occupational pollutants is presented.

Indexed as

acute kidney injuryacute tubular necrosischronic kidney diseaseoxidative stressreactive oxygen species

Identifiers

PMID37760075
PMCPMC10525550
OpenAlexW4386814128

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.