Evidence mapPaperPMID 37864030Full record

ReviewOncogene2023

Biophysics in tumor growth and progression: from single mechano-sensitive molecules to mechanomedicine.

Ying Xin, Keming Li, Miao Huang, Chenyu Liang, Dietmar Siemann, Lizi Wu, Youhua Tan, Xin Tang

Open access · hybridAbstract readReview
In one paragraph

Review in Oncogene, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 73 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
73citing papers in PubMed, 1 pooled it
18.8field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

73 citing papers in PubMed, 1 synthesis or guideline pooled it, 90 citations in OpenAlex.

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  20. Metabolic starvation-induced cell swelling drives solid stress in tumors.bioRxiv : the preprint server for biology · 2026
    Article

13 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 3 countries.

Ying Xin *The Hong Kong Polytechnic University Shenzhen Research Institute, Shenzhen, China.ORCID 0000-0002-9008-6105
Keming Li *The Hong Kong Polytechnic University Shenzhen Research Institute, Shenzhen, China.
Miao Huang *Department of Mechanical and Aerospace Engineering, Herbert Wertheim College of Engineering, University of Florida, Gainesville, FL, USA.
Chenyu Liang *Department of Mechanical and Aerospace Engineering, Herbert Wertheim College of Engineering, University of Florida, Gainesville, FL, USA.
Dietmar SiemannUF Health Cancer Center, University of Florida, Gainesville, FL, USA.
Lizi WuUF Health Cancer Center, University of Florida, Gainesville, FL, USA.ORCID 0000-0002-0076-2617
Youhua TanThe Hong Kong Polytechnic University Shenzhen Research Institute, Shenzhen, China. youhua.tan@polyu.edu.hk.
Xin TangDepartment of Mechanical and Aerospace Engineering, Herbert Wertheim College of Engineering, University of Florida, Gainesville, FL, USA. xin.tang@ufl.edu.ORCID 0000-0002-6206-4603
Hong Kong Polytechnic University · HKUniversity of Florida Health · USUniversity of Florida · US

Funding

Mechanisms and Functions of Unconventional Intercellular Calcium Waves in Electrically Non-excitable CellsR35GM150812 · UNIVERSITY OF FLORIDA · 2025 to 2025
$361k
NIGMS NIH HHS R35 GM150812
6 · The paper itself

Abstract

Evidence from physical sciences in oncology increasingly suggests that the interplay between the biophysical tumor microenvironment and genetic regulation has significant impact on tumor progression. Especially, tumor cells and the associated stromal cells not only alter their own cytoskeleton and physical properties but also remodel the microenvironment with anomalous physical properties. Together, these altered mechano-omics of tumor tissues and their constituents fundamentally shift the mechanotransduction paradigms in tumorous and stromal cells and activate oncogenic signaling within the neoplastic niche to facilitate tumor progression. However, current findings on tumor biophysics are limited, scattered, and often contradictory in multiple contexts. Systematic understanding of how biophysical cues influence tumor pathophysiology is still lacking. This review discusses recent different schools of findings in tumor biophysics that have arisen from multi-scale mechanobiology and the cutting-edge technologies. These findings range from the molecular and cellular to the whole tissue level and feature functional crosstalk between mechanotransduction and oncogenic signaling. We highlight the potential of these anomalous physical alterations as new therapeutic targets for cancer mechanomedicine. This framework reconciles opposing opinions in the field, proposes new directions for future cancer research, and conceptualizes novel mechanomedicine landscape to overcome the inherent shortcomings of conventional cancer diagnosis and therapies.

Indexed as

Mechanotransduction, CellularNeoplasmsBiophysicsHumansSignal TransductionTumor Microenvironment

Identifiers

PMID37864030
PMCPMC10656290
OpenAlexW4387817898

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.