Evidence map›Paper›PMID 37933137›Full record

ArticleClinical and applied thrombosis/hemostasis : official journal of the International Academy of Clinical and Applied Thrombosis/Hemostasis

The Correlation Between Cardiac Oxidative Stress and Inflammatory Cytokine Response Following Myocardial Infarction.

Dongpeng Duan, Tianfeng Fan, Linlin Zhang, Lingling Li, Honghu Wang, Mingming Guo, Yan Feng

Abstract read
In one paragraph

Article in Clinical and applied thrombosis/hemostasis : official journal of the International Academy of Clinical and Applied Thrombosis/Hemostasis. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
  4. Modulation of Redox-Sensitive Cardiac Ion Channels.Antioxidants (Basel, Switzerland) · 2025
    Review
  5. Article
  6. Article
  7. Cardioprotective effects ofFrontiers in pharmacology · 2025
    Article
  8. Review
  9. Review
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Dongpeng DuanDepartment of Emergency, Affiliated Hospital of Hebei Engineering University, Handan, China.
Tianfeng FanDepartment of Emergency, Affiliated Hospital of Hebei Engineering University, Handan, China.
Linlin ZhangDepartment of Emergency, Affiliated Hospital of Hebei Engineering University, Handan, China.
Lingling LiDepartment of Emergency, Affiliated Hospital of Hebei Engineering University, Handan, China.
Honghu WangDepartment of Emergency, Affiliated Hospital of Hebei Engineering University, Handan, China.
Mingming GuoDepartment of Emergency, Affiliated Hospital of Hebei Engineering University, Handan, China.
Yan FengDepartment of Critical Care Medicine, Affiliated Hospital of Hebei Engineering University, Handan, China.ORCID 0009-0003-5167-3416

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Our study was conducted to investigate the potential correlation between cardiac oxidative stress and inflammatory cytokine response following myocardial infarction. A total of 120 patients harboring acute myocardial infarction who underwent percutaneous coronary intervention (PCI) at our hospital were included. Their general clinical data were analyzed, and comparisons were made regarding the levels of inflammatory factors, oxidative stress markers, heart pump function, and cardiac function. The correlation between cardiac oxidative stress and inflammatory cytokine response was assessed using Pearson's linear correlation. Following treatment, significant reductions were seen in the serum levels of cortisol, thyroid-stimulating hormone (TSH), B-type natriuretic peptide (BNP), C-reactive protein (CRP), signal transducer and activator of transcription 3 (STAT3), interleukin-6 (IL-6), interleukin-8 (IL-8), tumor necrosis factor (TNF), and catalase (CAT) compared to pre-treatment levels. Conversely, the levels of growth hormone (GH), superoxide dismutase (SOD), glutathione peroxidase (GSH-Px), total antioxidant capacity (T-AOC), cardiac output (CO), and cardiac index (CI) were significantly elevated. Serum cortisol (

Indexed as

Myocardial InfarctionPercutaneous Coronary InterventionC-Reactive ProteinCytokinesHumansHydrocortisoneInterleukin-8Oxidative StressThyrotropinTumor Necrosis Factor-alphaC-Reactive ProteinCytokinesHydrocortisoneInterleukin-8ThyrotropinTumor Necrosis Factor-alphacardiac oxidative stresscorrelationinflammatory cytokinesmyocardial infarction

Identifiers

PMID37933137
PMCPMC10631341

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.