Evidence map›Paper›PMID 38064002›Full record

ReviewReviews in endocrine & metabolic disorders2024

Role of CaMKII in diabetes induced vascular injury and its interaction with anti-diabetes therapy.

Stephanie Chacar, Abdulhamid Abdi, Khalifa Almansoori, Jawaher Alshamsi, Cynthia Al Hageh, Pierre Zalloua, Ali A Khraibi, Stephen G Holt, Moni Nader

Open access · hybridAbstract readReview
In one paragraph

Review in Reviews in endocrine & metabolic disorders, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.8field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 12 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 1 institution in 1 country.

Stephanie ChacarDepartment of Physiology and Immunology, College of Medicine and Health Sciences, Khalifa University of Science and Technology, Abu Dhabi, United Arab Emirates. stefani.elsakar@ku.ac.ae.ORCID 0000-0003-4073-4749
Abdulhamid AbdiDepartment of Physiology and Immunology, College of Medicine and Health Sciences, Khalifa University of Science and Technology, Abu Dhabi, United Arab Emirates.ORCID 0000-0002-2159-5651
Khalifa AlmansooriDepartment of Physiology and Immunology, College of Medicine and Health Sciences, Khalifa University of Science and Technology, Abu Dhabi, United Arab Emirates.ORCID 0009-0003-4506-4788
Jawaher AlshamsiDepartment of Physiology and Immunology, College of Medicine and Health Sciences, Khalifa University of Science and Technology, Abu Dhabi, United Arab Emirates.ORCID 0009-0008-3191-9761
Cynthia Al HagehDepartment of Molecular Biology and Genetics, College of Medicine and Health Sciences, Khalifa University of Science and Technology, Abu Dhabi, United Arab Emirates.ORCID 0000-0001-8386-4063
Pierre ZallouaDepartment of Molecular Biology and Genetics, College of Medicine and Health Sciences, Khalifa University of Science and Technology, Abu Dhabi, United Arab Emirates.ORCID 0000-0002-8494-5081
Ali A KhraibiDepartment of Physiology and Immunology, College of Medicine and Health Sciences, Khalifa University of Science and Technology, Abu Dhabi, United Arab Emirates.ORCID 0000-0002-8002-2861
Stephen G HoltDepartment of Physiology and Immunology, College of Medicine and Health Sciences, Khalifa University of Science and Technology, Abu Dhabi, United Arab Emirates.ORCID 0000-0001-5004-5539
Moni NaderDepartment of Physiology and Immunology, College of Medicine and Health Sciences, Khalifa University of Science and Technology, Abu Dhabi, United Arab Emirates. moni.nader@ku.ac.ae.ORCID 0000-0003-2138-5213
Khalifa University of Science and Technology · AE

Funding

Khalifa University, United Arab Emirates FSU-2021-024
6 · The paper itself

Abstract

Diabetes mellitus is a metabolic disorder denoted by chronic hyperglycemia that drives maladaptive structural changes and functional damage to the vasculature. Attenuation of this pathological remodeling of blood vessels remains an unmet target owing to paucity of information on the metabolic signatures of this process. Ca

Indexed as

Diabetes MellitusVascular System InjuriesAnimalsCalcium-Calmodulin-Dependent Protein Kinase Type 2HumansSignal TransductionCalcium-Calmodulin-Dependent Protein Kinase Type 2CaMKIIDiabetesEndothelial cellsVascular smooth muscle cellsVasculature injury

Identifiers

PMID38064002
PMCPMC10943158
OpenAlexW4389484070

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.