Evidence map›Paper›PMID 38178684›Full record

ArticleCombinatorial chemistry & high throughput screening2024

Evaluation of the Mechanism of Yishan Formula in Treating Breast Cancer Based on Network Pharmacology and Experimental Verification.

Xiaoyue Lin, Wencheng Chi, Xue Geng, Qinghui Jiang, Baozhu Ma, Bowen Dai, Yutong Sui, Jiakang Jiang

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In one paragraph

Article in Combinatorial chemistry & high throughput screening, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.5field-weighted citation impact, top 39% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 2 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 2 institutions in 1 country.

Xiaoyue LinHeilongjiang University of Chinese Medicine, Harbin, 150000, China.
Wencheng ChiHeilongjiang University of Chinese Medicine, Harbin, 150000, China.
Xue GengHeilongjiang University of Chinese Medicine, Harbin, 150000, China.
Qinghui JiangHeilongjiang University of Chinese Medicine, Harbin, 150000, China.
Baozhu MaHeilongjiang University of Chinese Medicine, Harbin, 150000, China.
Bowen DaiHeilongjiang University of Chinese Medicine, Harbin, 150000, China.
Yutong SuiShenzhen Hospital of Southern Medical University, Shenzhen, 518110, China.
Jiakang JiangHeilongjiang University of Chinese Medicine, Harbin, 150000, China.
Heilongjiang University of Chinese Medicine · CNSouthern Medical University Shenzhen Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundYishan formula (YSF) has a significant effect on the treatment of breast cancer, which can improve the quality of life and prolong the survival of patients with breast cancer; however, its mechanism of action is unknown.

objectiveIn this study, network pharmacology and molecular docking methods have been used to explore the potential pharmacological effects of the YSF, and the predicted targets have been validated by in vitro experiments.

methodsActive components and targets of the YSF were obtained from the TCMSP and Swiss target prediction website. Four databases, namely GeneCards, OMIM, TTD, and DisGeNET, were used to search for disease targets. The Cytoscape v3.9.0 software was utilized to draw the network of drug-component-target and selected core targets. DAVID database was used to analyze the biological functions and pathways of key targets. Finally, molecular docking and in vitro experiments have been used to verify the hub genes.

resultsThrough data collection from the database, 157 active components and 618 genes implicated in breast cancer were obtained and treated using the YSF. After screening, the main active components (kaempferol, quercetin, isorhamnetin, dinatin, luteolin, and tamarixetin) and key genes (

conclusionThis study showed that resistance to breast cancer may be achieved by the synergy of multiple active components, target genes, and signal pathways, which can provide new avenues for breast cancer-targeted therapy.

Indexed as

Breast NeoplasmsDrugs, Chinese HerbalMolecular Docking SimulationNetwork PharmacologyAntineoplastic AgentsAntineoplastic Agents, PhytogenicCell ProliferationDrug Screening Assays, AntitumorFemaleHumansMCF-7 CellsAntineoplastic AgentsAntineoplastic Agents, PhytogenicDrugs, Chinese Herbalbreast cancerEGFR.experimental verificationIL6MAPK3molecular dockingnetwork pharmacologyYishan formula

Identifiers

PMID38178684
OpenAlexW4390609079

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.