Evidence map›Paper›PMID 38214284›Full record

ArticleJournal of the American Heart Association2024

Association Between Clonal Hematopoiesis Driver Mutations, Immune Cell Function, and the Vasculometabolic Complications of Obesity.

Helin Tercan, Benjamin C Cossins, Rosanne C van Deuren, Joost H W Rutten, Leo A B Joosten, Mihai G Netea, Alexander Hoischen, Siroon Bekkering, Niels P Riksen

Open access · goldAbstract read
In one paragraph

Article in Journal of the American Heart Association, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
1.8field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 4 citations in OpenAlex.

  1. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 3 countries.

Helin TercanRadboud University Medical Center Department of Internal Medicine Nijmegen the Netherlands.ORCID 0000-0002-5988-6147
Benjamin C CossinsRadboud University Medical Center Department of Internal Medicine Nijmegen the Netherlands.
Rosanne C van DeurenRadboud University Medical Center Department of Human Genetics Nijmegen the Netherlands.ORCID 0000-0003-1356-2611
Joost H W RuttenRadboud University Medical Center Department of Internal Medicine Nijmegen the Netherlands.ORCID 0000-0002-5259-9794
Leo A B JoostenRadboud University Medical Center Department of Internal Medicine Nijmegen the Netherlands.ORCID 0000-0001-6166-9830
Mihai G NeteaRadboud University Medical Center Department of Internal Medicine Nijmegen the Netherlands.
Alexander HoischenRadboud University Medical Center Department of Human Genetics Nijmegen the Netherlands.ORCID 0000-0002-8072-4476
Siroon BekkeringRadboud University Medical Center Department of Internal Medicine Nijmegen the Netherlands.ORCID 0000-0003-1149-466X
Niels P RiksenRadboud University Medical Center Department of Internal Medicine Nijmegen the Netherlands.ORCID 0000-0001-9197-8124
Radboud University Nijmegen · NLUniversity of Bonn · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundObesity is accompanied by dysregulated inflammation, which can contribute to vasculometabolic complications including metabolic syndrome and atherosclerosis. Recently, clonal hematopoiesis of indeterminate potential (CHIP) has emerged as a risk factor for cardiovascular diseases. We aimed to determine how CHIP is related to immune cell function, systemic inflammation, and vasculometabolic complications in obese individuals. METHODS AND

resultsTwo hundred ninety-seven individuals with overweight and obesity, between the ages of 54 and 81 years, were recruited in a cross-sectional study. Clonal hematopoiesis driver mutations (CHDMs) were identified with an ultrasensitive targeted assay. Assessment of carotid artery atherosclerosis was performed with ultrasound. Detailed immunological parameters, including cytokine production capacity of peripheral blood mononuclear cells, and targeted plasma proteomics analysis, were studied. Adipose tissue inflammation was determined in subcutaneous fat biopsies. Individuals with CHIP had higher concentrations of circulating IL (interleukin)-6. Total number of leukocytes and neutrophils were higher in individuals with CHIP. In contrast, ex vivo cytokine production capacity of peripheral blood mononuclear cells was significantly lower in individuals with CHIP. Sex-stratified analysis showed that men with CHDMs had significantly higher leukocyte and neutrophil counts, and ex vivo cytokine production capacity was lower in women with CHDMs. Surprisingly, the presence of atherosclerotic plaques was significantly lower in individuals with CHDMs. There was no relation between CHIP and metabolic syndrome.

conclusionsIn individuals with overweight or obesity, CHDMs are not associated with vasculometabolic complications, but rather with a lower presence of carotid plaques. CHDMs associate with increased circulating inflammatory markers and leukocyte numbers, but a lower peripheral blood mononuclear cell cytokine production capacity.

Indexed as

AtherosclerosisMetabolic SyndromeAgedAged, 80 and overClonal HematopoiesisCross-Sectional StudiesFemaleHematopoiesisHumansInflammationInterleukin-6Leukocytes, MononuclearMaleMiddle AgedMutationObesityInterleukin-6atherosclerosisclonal hematopoiesisimmunological parametersobesity

Identifiers

PMID38214284
PMCPMC10926828
OpenAlexW4390792714

What Socratic holds

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LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.