Evidence mapPaperPMID 38313232Full record

ArticleWorld journal of gastroenterology2024

Circulating microRNA expression and nonalcoholic fatty liver disease in adolescents with severe obesity.

Yi-Jie Li, Brittney O Baumert, Nikos Stratakis, Jesse A Goodrich, Hao-Tian Wu, Jing-Xuan He, Yin-Qi Zhao, Max T Aung, Hong-Xu Wang, Sandrah P Eckel and 13 more

Open access · hybridAbstract read
In one paragraph

Article in World journal of gastroenterology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
16citing papers in PubMed, 1 pooled it
6.2field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

16 citing papers in PubMed, 1 synthesis or guideline pooled it, 16 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

23 authors at 7 institutions in 1 country.

Yi-Jie LiDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States.
Brittney O BaumertDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States.
Nikos StratakisBarcelona Institute of Global Health, Barcelona Institute of Global Health, Barcelona 08036, Spain.
Jesse A GoodrichDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States.
Hao-Tian WuDepartment of Environmental Health Sciences, Mailman School of Public Health, Columbia University, New York, NY 10032, United States.
Jing-Xuan HeDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States.
Yin-Qi ZhaoDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States.
Max T AungDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States.
Hong-Xu WangDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States.
Sandrah P EckelDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States.
Douglas I WalkerGangarosa Department of Environmental Health, Rollins School of Public Health, Emory University, Atlanta, GA 30329, United States.
Damaskini ValviDepartment of Environmental Medicine and Public Health, Icahn School of Medicine at Mount Sinai, New York, NY 10029, United States.
Michele A La MerrillDepartment of Environmental Toxicology, University of California, Davis, CA 95616, United States.
Justin R RyderDepartment of Surgery, Lurie Children's Hospital of Chicago, Chicago, IL 60611, United States.
Thomas H IngeDepartment of Surgery, Lurie Children's Hospital of Chicago, Chicago, IL 60611, United States.
Todd JenkinsDivision of Biostatistics and Epidemiology, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, United States.
Stephanie SisleyDepartment of Pediatrics, Children's Nutrition Research Center USDA/ARS, Baylor College of Medicine, Houston, TX 77030, United States.
Rohit KohliDepartment of Gastroenterology, Children's Hospital Los Angeles, Los Angeles, CA 90027, United States.
Stavra A XanthakosDepartment of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH 45229, United States.
Andrea A BaccarelliDepartment of Environmental Health Sciences, Mailman School of Public Health, Columbia University, New York, NY 10032, United States.
Rob McConnellDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States.
David V ContiDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States.
Lida ChatziDepartment of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA 90032, United States. chatzi@usc.edu.
University of Southern California · USColumbia University · USCincinnati Children's Hospital Medical Center · USEmory University · USIcahn School of Medicine at Mount Sinai · USUniversity of California, Davis · USUniversity of Cincinnati Medical Center · US

Funding

Pilot Projects ProgramP30ES007048 · UNIVERSITY OF SOUTHERN CALIFORNIA · 1996 to 2025
$10.4M
Statistical Methods for Integrative Genomics in CancerP01CA196569 · UNIVERSITY OF SOUTHERN CALIFORNIA · 2025 to 2025
$2.5M
Longitudinal integration of environmental exposures, omics, and childhood NAFLD (LEON) StudyU01HG013288 · UNIVERSITY OF SOUTHERN CALIFORNIA · 2025 to 2025
$784k
Brain VDR Regulate Glucose BalanceR01DK128117 · BAYLOR COLLEGE OF MEDICINE · 2025 to 2025
$497k
NCI NIH HHS P01 CA196569NHGRI NIH HHS U01 HG013288NIDDK NIH HHS R01 DK128117NIDDK NIH HHS UM1 DK072493NIDDK NIH HHS UM1 DK095710NIEHS NIH HHS P30 ES007048NIEHS NIH HHS R01 ES030691
6 · The paper itself

Abstract

backgroundNonalcoholic fatty liver disease (NAFLD) is one of the most common chronic liver diseases in children and adolescents. NAFLD ranges in severity from isolated hepatic steatosis to nonalcoholic steatohepatitis (NASH), wherein hepatocellular inflammation and/or fibrosis coexist with steatosis. Circulating microRNA (miRNA) levels have been suggested to be altered in NAFLD, but the extent to which miRNA are related to NAFLD features remains unknown. This analysis tested the hypothesis that plasma miRNAs are significantly associated with histological features of NAFLD in adolescents.

aimTo investigate the relationship between plasma miRNA expression and NAFLD features among adolescents with NAFLD.

methodsThis study included 81 adolescents diagnosed with NAFLD and 54 adolescents without NAFLD from the Teen-Longitudinal Assessment of Bariatric Surgery study. Intra-operative core liver biopsies were collected from participants and used to characterize histological features of NAFLD. Plasma samples were collected during surgery for miRNA profiling. A total of 843 plasma miRNAs were profiled using the HTG EdgeSeq platform. We examined associations of plasma miRNAs and NAFLD features using logistic regression after adjusting for age, sex, race, and other key covariates. Ingenuity Pathways Analysis was used to identify biological functions of miRNAs that were associated with multiple histological features of NAFLD.

resultsWe identified 16 upregulated plasma miRNAs, including miR-193a-5p and miR-193b-5p, and 22 downregulated plasma miRNAs, including miR-1282 and miR-6734-5p, in adolescents with NAFLD. Moreover, 52, 16, 15, and 9 plasma miRNAs were associated with NASH, fibrosis, ballooning degeneration, and lobular inflammation, respectively. Collectively, 16 miRNAs were associated with two or more histological features of NAFLD. Among those miRNAs, miR-411-5p was downregulated in NASH, ballooning, and fibrosis, while miR-122-5p, miR-1343-5p, miR-193a-5p, miR-193b-5p, and miR-7845-5p were consistently and positively associated with all histological features of NAFLD. Pathway analysis revealed that most common pathways of miRNAs associated with multiple NAFLD features have been associated with tumor progression, while we also identified linkages between miR-122-5p and hepatitis C virus and between miR-199b-5p and chronic hepatitis B.

conclusionPlasma miRNAs were associated with NAFLD features in adolescent with severe obesity. Larger studies with more heterogeneous NAFLD phenotypes are needed to evaluate miRNAs as potential biomarkers of NAFLD.

Indexed as

Circulating MicroRNAMicroRNAsNon-alcoholic Fatty Liver DiseaseObesity, MorbidAdolescentChildFibrosisHumansInflammationLiverObesityCirculating MicroRNAMicroRNAsMIRN411 microRNA, humanBallooning degenerationLiver fibrosisLobular inflammationMicroRNANonalcoholic fatty liver diseaseNon-alcoholic steatohepatitis

Identifiers

PMID38313232
PMCPMC10835537
OpenAlexW4391241465

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.