Evidence map›Paper›PMID 38338673›Full record

ArticleInternational journal of molecular sciences2024

A Missense Variant in

Ricardo Usategui-Martín, Nadia Galindo-Cabello, Salvador Pastor-Idoate, José María Fernández-Gómez, Álvaro Del Real, Diego Ferreño, Rebeca Lapresa, Francisco Martín-Rodriguez, José A Riancho, Ángeles Almeida and 1 more

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
0.6field-weighted citation impact, top 32% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 2 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 3 institutions in 1 country.

Ricardo Usategui-MartínDepartment of Cell Biology, Genetics, Histology and Pharmacology, Faculty of Medicine, University of Valladolid, 47003 Valladolid, Spain.ORCID 0000-0001-7699-4388
Nadia Galindo-CabelloDepartment of Cell Biology, Genetics, Histology and Pharmacology, Faculty of Medicine, University of Valladolid, 47003 Valladolid, Spain.ORCID 0000-0001-8721-5220
Salvador Pastor-IdoateIOBA-Eye Institute, University of Valladolid, 47011 Valladolid, Spain.ORCID 0000-0002-6463-4227
José María Fernández-GómezDepartment of Cell Biology, Genetics, Histology and Pharmacology, Faculty of Medicine, University of Valladolid, 47003 Valladolid, Spain.
Álvaro Del RealDepartment of Medicine and Psychiatry, Faculty of Medicine, Valdecilla Research Institute (IDIVAL), University of Cantabria, 39011 Santander, Spain.ORCID 0000-0002-1057-461X
Diego FerreñoLaboratory of the Materials Science and Engineering Division-LADICIM, Faculty of Civil Engineering, University of Cantabria, 39011 Santander, Spain.ORCID 0000-0003-3533-1881
Rebeca LapresaInstitute of Functional Biology and Genomics, University of Salamanca, CSIC, 37008 Salamanca, Spain.ORCID 0000-0001-8901-3972
Francisco Martín-RodriguezDepartment of Medicine, Dermatology and Toxicology, Faculty of Medicine, University of Valladolid, 47003 Valladolid, Spain.
José A RianchoDepartment of Medicine and Psychiatry, Faculty of Medicine, Valdecilla Research Institute (IDIVAL), University of Cantabria, 39011 Santander, Spain.
Ángeles AlmeidaInstitute of Functional Biology and Genomics, University of Salamanca, CSIC, 37008 Salamanca, Spain.ORCID 0000-0003-0485-8904
José Luis Pérez-CastrillónDepartment of Medicine, Dermatology and Toxicology, Faculty of Medicine, University of Valladolid, 47003 Valladolid, Spain.
Universidad de Valladolid · ESUniversidad de Cantabria · ESUniversidad de Salamanca · ES

Funding

Instituto de Salud Carlos III PI21/00727Ministerio de Ciencia e Innovación PID2020-114585RA-I00
6 · The paper itself

Abstract

Metabolic bone diseases cover a broad spectrum of disorders that share alterations in bone metabolism that lead to a defective skeleton, which is associated with increasing morbidity, disability, and mortality. There is a close connection between the etiology of metabolic bone diseases and genetic factors, with

Indexed as

Bone Diseases, MetabolicTumor Suppressor Protein p53AnimalsBiomarkersBone and BonesCase-Control StudiesGenetic Predisposition to DiseaseHumansMicePolymorphism, Single NucleotideBiomarkersTumor Suppressor Protein p53apoptosis and gene polymorphismmetabolic bone diseasesosteoporosisp53TP53

Identifiers

PMID38338673
PMCPMC10855390
OpenAlexW4391170523

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.