Evidence map›Paper›PMID 38358463›Full record

ArticleJournal of endocrinological investigation2024

Serum levels of adiponectin differentiate generalized lipodystrophies from anorexia nervosa.

G Ceccarini, C Pelosini, M Paoli, N Tyutyusheva, S Magno, D Gilio, L Palladino, M R Sessa, S Bertelloni, F Santini

Abstract read
PubMed Publisher
In one paragraph

Article in Journal of endocrinological investigation, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
1.6field-weighted citation impact, top 19% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 6 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Review
  5. SIRT1 Serum Concentrations in Lipodystrophic Syndromes.International journal of molecular sciences · 2024
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 1 country.

G CeccariniObesity and Lipodystrophy Center, Endocrinology Unit, University Hospital of Pisa, Pisa, Italy. giovanni.ceccarini@unipi.it.ORCID http://orcid.org/0000-0003-0701-642X
C PelosiniObesity and Lipodystrophy Center, Endocrinology Unit, University Hospital of Pisa, Pisa, Italy.
M PaoliChemistry and Endocrinology Laboratory, University Hospital of Pisa, Pisa, Italy.
N TyutyushevaPediatric Unit, University Hospital of Pisa, Pisa, Italy.
S MagnoObesity and Lipodystrophy Center, Endocrinology Unit, University Hospital of Pisa, Pisa, Italy.
D GilioObesity and Lipodystrophy Center, Endocrinology Unit, University Hospital of Pisa, Pisa, Italy.
L PalladinoObesity and Lipodystrophy Center, Endocrinology Unit, University Hospital of Pisa, Pisa, Italy.
M R SessaChemistry and Endocrinology Laboratory, University Hospital of Pisa, Pisa, Italy.
S BertelloniPediatric Unit, University Hospital of Pisa, Pisa, Italy.
F SantiniObesity and Lipodystrophy Center, Endocrinology Unit, University Hospital of Pisa, Pisa, Italy.
Azienda Ospedaliera Universitaria Pisana · ITUniversity of Pisa · IT

Funding

Ministero dell'Università e della Ricerca PRIN 2020NCKXBR
6 · The paper itself

Abstract

purposeThe differential diagnosis of lipodystrophy involves other disorders characterized by severe fat loss and may be sometimes challenging. Owing to the rarity of lipodystrophy, it is relevant to search for tools and assays that differentiate it from other diseases that may mimic it. We conducted a study on leptin and high molecular weight (HMW) adiponectin serum concentrations in a series of patients diagnosed with lipodystrophy and compared them with those found in anorexia nervosa, one of the illnesses that may be cause of a missed diagnosis of lipodystrophy.

methodsLeptin and HMW adiponectin serum concentrations were measured in six patients diagnosed with generalized lipodystrophy (GL), six with progeroid syndromes (PS), 13 with familial partial lipodystrophy type 1 (FPLD1, Kobberling syndrome), 10 with familial partial lipodystrophy type 2 (FPLD2, Dunnigan syndrome), 18 with acquired partial lipodystrophy (APL) and 12 affected by anorexia nervosa (AN). Measurements were compared to those obtained in 12 normal weight healthy subjects.

resultsSerum leptin concentrations were reduced to a similar degree in GL, PS and AN, proportionally to the extent of fat loss. Serum concentrations of HMW adiponectin were found extremely low in patients with GL and PS, while comparable to normal weight subjects in patients with AN.

conclusionSerum HMW adiponectin can be regarded as a useful tool to discriminate between generalized lipodystrophy syndromes (including PS) and AN.

Indexed as

AdiponectinAnorexia NervosaLeptinAdolescentAdultBiomarkersCase-Control StudiesChildDiagnosis, DifferentialFemaleHumansLipodystrophyLipodystrophy, Congenital GeneralizedMaleMiddle AgedYoung AdultAdiponectinADIPOQ protein, humanBiomarkersLeptinAnorexia nervosaHigh molecular weight adiponectinLeptinLipodystrophyProgeroid lipodystrophies

Identifiers

PMID38358463
OpenAlexW4391847077

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.