Evidence map›Paper›PMID 38390204›Full record

ReviewFrontiers in endocrinology2024

LncRNA as a regulator in the development of diabetic complications.

Mengrou Geng, Wei Liu, Jinjie Li, Ge Yang, Yuan Tian, Xin Jiang, Ying Xin

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in endocrinology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed, 1 pooled it
4.6field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 1 synthesis or guideline pooled it, 16 citations in OpenAlex.

  1. Pooled it
  2. Review
  3. Review
  4. Article
  5. Article
  6. Observational
  7. Article
  8. Curcumol ameliorates diabetic retinopathyWorld journal of diabetes · 2025
    Article
  9. Article
  10. Genetic/epigenetic RNA dysregulation in type 2Frontiers in endocrinology · 2025
    Article
  11. Article
  12. Article
  13. The mFrontiers in endocrinology · 2024
    Review
  14. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Mengrou GengJilin Provincial Key Laboratory of Radiation Oncology & Therapy, The First Hospital of Jilin University and College of Basic Medical Science, Jilin University, Changchun, China.
Wei LiuKey Laboratory of Pathobiology, Ministry of Education, College of Basic Medical Science, Jilin University, Changchun, China.
Jinjie LiKey Laboratory of Pathobiology, Ministry of Education, College of Basic Medical Science, Jilin University, Changchun, China.
Ge YangKey Laboratory of Pathobiology, Ministry of Education, College of Basic Medical Science, Jilin University, Changchun, China.
Yuan TianKey Laboratory of Pathobiology, Ministry of Education, College of Basic Medical Science, Jilin University, Changchun, China.
Xin JiangJilin Provincial Key Laboratory of Radiation Oncology & Therapy, The First Hospital of Jilin University and College of Basic Medical Science, Jilin University, Changchun, China.
Ying XinKey Laboratory of Pathobiology, Ministry of Education, College of Basic Medical Science, Jilin University, Changchun, China.
Jilin University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Diabetes is a metabolic disease characterized by hyperglycemia, which induces the production of AGEs, ROS, inflammatory cytokines, and growth factors, leading to the formation of vascular dysfunction and target organ damage, promoting the development of diabetic complications. Diabetic nephropathy, retinopathy, and cardiomyopathy are common complications of diabetes, which are major contributors to disability and death in people with diabetes. Long non-coding RNAs affect gene transcription, mRNA stability, and translation efficiency to influence gene expression for a variety of biological functions. Over the past decade, it has been demonstrated that dysregulated long non-coding RNAs are extensively engaged in the pathogenesis of many diseases, including diabetic complications. Thus, this review discusses the regulations of long non-coding RNAs on the primary pathogenesis of diabetic complications (oxidative stress, inflammation, fibrosis, and microvascular dysfunction), and some of these long non-coding RNAs may function as potential biomarkers or therapeutic targets for diabetic complications.

Indexed as

Diabetes ComplicationsDiabetes MellitusDiabetic NephropathiesHyperglycemiaRNA, Long NoncodingCytokinesHumansCytokinesRNA, Long Noncodingdiabetic cardiomyopathydiabetic nephropathydiabetic retinopathylong non-coding RNAsmicroRNA

Identifiers

PMID38390204
PMCPMC10881719
OpenAlexW4391648620

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.