Evidence mapPaperPMID 38520487Full record

ArticleMolecular biology reports2024

Quercetin declines LPS induced inflammation and augments adiponectin expression in 3T3-L1 differentiated adipocytes SIRT-1 dependently.

Zahra Noori, Mohammad Sharifi, Sanaz Dastghaib, Fatemeh Basiri Kejani, Fatemeh Roohy, Zahra Ansari, Mohammad Hasan Maleki, Morvarid Siri, Sayed Mohammad Shafiee

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Article in Molecular biology reports, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
2.1field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 6 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 4 institutions in 1 country.

Zahra Noori *Department of Anatomical sciences, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Mohammad Sharifi *Department of Genetics, Faculty of Advanced Science and Technology, Tehran Medical Sciences, Islamic Azad University, Tehran, Iran.ORCID http://orcid.org/0009-0004-9899-7968
Sanaz DastghaibEndocrinology and Metabolism Research Center, Shiraz University of Medical Science, Shiraz, Iran.ORCID http://orcid.org/0000-0001-8553-9221
Fatemeh Basiri KejaniDepartment of Medical Nanotechnology, School of advanced sciences and technologies, Shiraz University of Medical Sciences, Shiraz, Iran.ORCID http://orcid.org/0009-0009-0911-0680
Fatemeh RoohyDepartment of Genetics, Islamic Azad University, Kazerun, Iran.ORCID http://orcid.org/0009-0009-1320-0574
Zahra AnsariDepartment of Genetics, Faculty of Biological Sciences and Technology, Shahid Ashrafi Esfahani university, Esfahan, Iran.
Mohammad Hasan MalekiDepartment of Clinical Biochemistry, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.ORCID http://orcid.org/0000-0002-0095-4264
Morvarid SiriAutophagy Research Centre, Department of Clinical Biochemistry, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.ORCID http://orcid.org/0000-0002-5556-2102
Sayed Mohammad ShafieeAutophagy Research Centre, Department of Clinical Biochemistry, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran. shafieem@sums.ac.ir.ORCID http://orcid.org/0000-0002-9221-2267
Shiraz University of Medical Sciences · IRIslamic Azad University Kazeron · IRIslamic Azad University Medical Branch of Tehran · IRShiraz University · IR

Funding

Shiraz University of Medical Sciences 28753
6 · The paper itself

Abstract

backgroundInflammation is an important factor contributing to obesity-induced metabolic disorders. Different investigations confirm that local inflammation in adipose issues is the primary reason for such disorder, resulting in low-grade systemic inflammation. Anti-inflammatory, antioxidant, and epigenetic modification are among the varied properties of Quercetin (QCT) as a natural flavonoid.

objectiveThe precise molecular mechanism followed by QCT to alleviate inflammation has been unclear. This study explores whether the anti-inflammatory effects of QCT in 3T3-L1 differentiated adipocytes may rely on SIRT-1.

methodsThe authors isolated 3T3-L1 pre-adipocyte cells and exposed them to varying concentrations of QCT, lipopolysaccharide (LPS), and a selective inhibitor of silent mating type information regulation 2 homolog 1 (SIRT-1) called EX-527. After determining the optimal dosages of QCT, LPS, and EX-527, they assessed the mRNA expression levels of IL-18, IL-1, IL-6, TNF-α, SIRT-1, and adiponectin using quantitative reverse transcription-polymerase chain reaction (qRT-PCR).

resultsThe study showed considerable cytotoxic effects of LPS (200 ng/mL) + QCT (100 µM) + EX-527 (10 µM) on 3T3-L1 differentiated adipocytes after 48 h of incubation. QCT significantly upregulated the expression levels of adiponectin and SIRT-1 (p < 0.0001). However, introducing SIRT-1 inhibitor (p < 0.0001) reversed the impact of QCT on adiponectin expression. Additionally, QCT reduced SIRT-1-dependent pro-inflammatory cytokines in 3T3-L1 differentiated adipocytes (p < 0.0001).

conclusionThis study revealed that QCT treatment reduced crucial pro-inflammatory cytokines levels and increased adiponectin levels following LPS treatment. This finding implies that SIRT-1 may be a crucial factor for the anti-inflammatory activity of QCT.

Indexed as

AdiponectinLipopolysaccharidesQuercetinSirtuin 13T3-L1 CellsAdipocytesAnimalsAnti-Inflammatory AgentsCytokinesInflammationMiceAdiponectinAnti-Inflammatory AgentsCytokinesLipopolysaccharidesQuercetinSirt1 protein, mouseSirtuin 1

Identifiers

PMID38520487
OpenAlexW4393111685

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.