Evidence mapPaperPMID 38530777Full record

Observational studyJournal of the National Cancer Institute2024

Metformin boosts antitumor immunity and improves prognosis in upfront resected pancreatic cancer: an observational study.

Casper W F van Eijck, Disha Vadgama, Casper H J van Eijck, Johanna W Wilmink, Dutch Pancreatic Cancer Group (DPCG)

Open access · hybridAbstract readObservational Study
In one paragraph

Observational study in Journal of the National Cancer Institute, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 23 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
23citing papers in PubMed, 1 pooled it
7.9field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

23 citing papers in PubMed, 1 synthesis or guideline pooled it, 28 citations in OpenAlex.

  1. Pooled it
  2. p53 and p21 Status Influences Cellular Response to Metformin inCurrent issues in molecular biology · 2026
    Article
  3. A Phase 2 Feasibility Study Combining Pembrolizumab and Metformin in patients with Metastatic Head and Neck Cancer.Clinical cancer research : an official journal of the American Association for Cancer Research · 2026
    Article
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  13. Unlocking Novel Therapeutic Potential of Angiotensin II Receptor Blockers.International journal of molecular sciences · 2025
    Review
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  20. Extrinsic and Cell-Intrinsic Stress in the Immune Tumor Micro-Environment.International journal of molecular sciences · 2024
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Casper W F van EijckDepartment of Surgery, Erasmus University Medical Centre, Rotterdam, the Netherlands.
Disha VadgamaErasmus MC Cancer Institute, Erasmus University Medical Centre, Rotterdam, the Netherlands.
Casper H J van EijckDepartment of Surgery, Erasmus University Medical Centre, Rotterdam, the Netherlands.ORCID 0000-0002-9511-2157
Johanna W WilminkDepartment of Medical Oncology, Amsterdam University Medical Centre, Amsterdam, the Netherlands.
Dutch Pancreatic Cancer Group (DPCG)
Erasmus MC Cancer Institute · NL

Funding

Survival With Pancreatic Cancer Foundation OVIT17-06
6 · The paper itself

Abstract

backgroundBeyond demographic and immune factors, metabolic considerations, particularly metformin's recognized impact in oncology, warrant exploration in treating pancreatic cancer. This study aimed to investigate the influence of metformin on patient survival and its potential correlation with distinct immune profiles in pancreatic ductal adenocarcinoma (PDAC) tumors.

methodsWe included 82 upfront resected and 66 gemcitabine-based neoadjuvant chemoradiotherapy (nCRT)-treated patients from the PREOPANC randomized controlled trial (RCT). Transcriptomic NanoString immunoprofiling was performed for a subset of 96 available resected specimens.

resultsDisparities in survival outcomes and immune profiles were apparent between metformin and non-metformin users in upfront resected patients but lacking in nCRT-treated patients. Compared to non-metformin users, upfront resected metformin users showed a higher median overall survival (OS) of 29 vs 14 months and a better 5-year OS rate of 19% vs 5%. Furthermore, metformin use was a favorable prognostic factor for OS in the upfront surgery group (HR = 0.56; 95% CI = 0.32 to 0.99). Transcriptomic data revealed that metformin users significantly underexpressed genes related to pro-tumoral immunity, including monocyte to M2 macrophage polarization and activation. Furthermore, the relative abundance of anti-inflammatory CD163+ MRC1+ M2 macrophages in non-metformin users and immune-activating CD1A+ CD1C+ dendritic cells in metformin users was heightened (P < .001).

conclusionThis study unveils immune profile changes resulting from metformin use in upfront resected pancreatic cancer patients, possibly contributing to prolonged survival outcomes. Specifically, metformin use may decrease the abundance and activity of pro-tumoral M2 macrophages and increase the recruitment and function of tumor-resolving DCs, favoring antitumor immunity.[PREOPANC trial EudraCT: 2012-003181-40].

Indexed as

Carcinoma, Pancreatic DuctalMetforminPancreatic NeoplasmsAgedDeoxycytidineFemaleGemcitabineHumansHypoglycemic AgentsMaleMiddle AgedNeoadjuvant TherapyPrognosisRandomized Controlled Trials as TopicDeoxycytidineGemcitabineHypoglycemic AgentsMetformin

Identifiers

PMID38530777
PMCPMC11308183
OpenAlexW4393185973

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.