Evidence mapPaperPMID 38548778Full record

ArticleScientific reports2024

Cyclosporine-induced kidney damage was halted by sitagliptin and hesperidin via increasing Nrf2 and suppressing TNF-α, NF-κB, and Bax.

Ahmed M Abd-Eldayem, Sohayla Mahmoud Makram, Basim Anwar Shehata Messiha, Hanan H Abd-Elhafeez, Mustafa Ahmed Abdel-Reheim

Open access · goldAbstract read
In one paragraph

Article in Scientific reports, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.

0numbers the graph read from it
0cells of the map it votes in
16citing papers in PubMed
14.2field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

16 citing papers in PubMed, 34 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 3 institutions in 2 countries.

Ahmed M Abd-EldayemDepartment of Medical Pharmacology, Faculty of Medicine, Assiut University, Assiut, Egypt. dr.ahmed2016@aun.edu.eg.
Sohayla Mahmoud MakramDepartment of Pharmacology, Faculty of Medicine, Merit University, Sohâg, Egypt.
Basim Anwar Shehata MessihaDepartment of Pharmacology and Toxicology, Faculty of Pharmacy, Beni-Suef University, Beni Suef, Egypt.
Hanan H Abd-ElhafeezDepartment of Cell and Tissue, Faculty of Veterinary Medicine, Assiut University, Assiut, Egypt.
Mustafa Ahmed Abdel-ReheimDepartment of Pharmacology and Toxicology, Faculty of Pharmacy, Beni-Suef University, Beni Suef, Egypt.
Assiut University · EGBeni-Suef University · EGSohag University · EG

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cyclosporine A (CsA) is employed for organ transplantation and autoimmune disorders. Nephrotoxicity is a serious side effect that hampers the therapeutic use of CsA. Hesperidin and sitagliptin were investigated for their antioxidant, anti-inflammatory, and tissue-protective properties. We aimed to investigate and compare the possible nephroprotective effects of hesperidin and sitagliptin. Male Wistar rats were utilized for induction of CsA nephrotoxicity (20 mg/kg/day, intraperitoneally for 7 days). Animals were treated with sitagliptin (10 mg/kg/day, orally for 14 days) or hesperidin (200 mg/kg/day, orally for 14 days). Blood urea, serum creatinine, albumin, cystatin-C (CYS-C), myeloperoxidase (MPO), and glucose were measured. The renal malondialdehyde (MDA), glutathione (GSH), catalase, and SOD were estimated. Renal TNF-α protein expression was evaluated. Histopathological examination and immunostaining study of Bax, Nrf-2, and NF-κB were performed. Sitagliptin or hesperidin attenuated CsA-mediated elevations of blood urea, serum creatinine, CYS-C, glucose, renal MDA, and MPO, and preserved the serum albumin, renal catalase, SOD, and GSH. They reduced the expressions of TNF-α, Bax, NF-κB, and pathological kidney damage. Nrf2 expression in the kidney was raised. Hesperidin or sitagliptin could protect the kidney against CsA through the mitigation of oxidative stress, apoptosis, and inflammation. Sitagliptin proved to be more beneficial than hesperidin.

Indexed as

HesperidinKidney DiseasesRenal InsufficiencyAnimalsbcl-2-Associated X ProteinCatalaseCreatinineCyclosporineGlucoseGlutathioneKidneyMaleNF-E2-Related Factor 2NF-kappa BOxidative StressRatsbcl-2-Associated X ProteinCatalaseCreatinineCyclosporineGlucoseGlutathioneHesperidinNF-E2-Related Factor 2NF-kappa BSitagliptin PhosphateSuperoxide DismutaseTumor Necrosis Factor-alphaUreaCyclosporineHesperidinNephrotoxicityNF-κBNrf2Sitagliptin

Identifiers

PMID38548778
PMCPMC10978894
OpenAlexW4393279693

What Socratic holds

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LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.