Evidence mapPaperPMID 38617433Full record

ReviewMedComm2024

Inflammation in diabetes complications: molecular mechanisms and therapeutic interventions.

Lu Zhao, Haoran Hu, Lin Zhang, Zheting Liu, Yunchao Huang, Qian Liu, Liang Jin, Meifei Zhu, Ling Zhang

Open access · goldAbstract readReview
In one paragraph

Review in MedComm, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 70 papers, 3 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
70citing papers in PubMed, 3 pooled it
35.6field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

70 citing papers in PubMed, 3 syntheses or guidelines pooled it, 93 citations in OpenAlex.

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  20. Multi-Omics Analysis of a Spontaneous Type 2 Diabetes Model inInternational journal of molecular sciences · 2026
    Article

10 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 1 institution in 2 countries.

Lu ZhaoDepartment of Biology and Medicine College of Life Science, Zhejiang Chinese Medical University Hangzhou China.
Haoran HuDepartment of Biology and Medicine College of Life Science, Zhejiang Chinese Medical University Hangzhou China.
Lin ZhangDepartment of Biology and Medicine College of Life Science, Zhejiang Chinese Medical University Hangzhou China.
Zheting LiuDepartment of Biology and Medicine College of Life Science, Zhejiang Chinese Medical University Hangzhou China.
Yunchao HuangDepartment of Biology and Medicine College of Life Science, Zhejiang Chinese Medical University Hangzhou China.
Qian LiuNational Demonstration Center for Experimental Traditional Chinese Medicines Education (Zhejiang Chinese Medical University) College of Pharmaceutical Science, Zhejiang Chinese Medical University Hangzhou China.
Liang JinDepartment of Biology and Medicine College of Life Science, Zhejiang Chinese Medical University Hangzhou China.
Meifei ZhuDepartment of Critical Care Medicine The First Affiliated Hospital of Zhejiang Chinese Medical University (Zhejiang Provincial Hospital of Chinese Medicine) Hangzhou China.
Ling ZhangDepartment of Biology and Medicine College of Life Science, Zhejiang Chinese Medical University Hangzhou China.ORCID https://orcid.org/0000-0001-7542-1879
Zhejiang Chinese Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

At present, diabetes mellitus (DM) has been one of the most endangering healthy diseases. Current therapies contain controlling high blood sugar, reducing risk factors like obesity, hypertension, and so on; however, DM patients inevitably and eventually progress into different types of diabetes complications, resulting in poor quality of life. Unfortunately, the clear etiology and pathogenesis of diabetes complications have not been elucidated owing to intricate whole-body systems. The immune system was responsible to regulate homeostasis by triggering or resolving inflammatory response, indicating it may be necessary to diabetes complications. In fact, previous studies have been shown inflammation plays multifunctional roles in the pathogenesis of diabetes complications and is attracting attention to be the meaningful therapeutic strategy. To this end, this review systematically concluded the current studies over the relationships of susceptible diabetes complications (e.g., diabetic cardiomyopathy, diabetic retinopathy, diabetic peripheral neuropathy, and diabetic nephropathy) and inflammation, ranging from immune cell response, cytokines interaction to pathomechanism of organ injury. Besides, we also summarized various therapeutic strategies to improve diabetes complications by target inflammation from special remedies to conventional lifestyle changes. This review will offer a panoramic insight into the mechanisms of diabetes complications from an inflammatory perspective and also discuss contemporary clinical interventions.

Indexed as

diabetes complicationsinflammationmolecular mechanismstherapeutic interventions

Identifiers

PMID38617433
PMCPMC11014467
OpenAlexW4394743252

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.