Evidence map›Paper›PMID 38702316›Full record

ArticleNature communications2024

LncRNA-LncDACH1 mediated phenotypic switching of smooth muscle cells during neointimal hyperplasia in male arteriovenous fistulas.

Zhaozheng Li, Yao Zhao, Zhenwei Pan, Benzhi Cai, Chengwei Zhang, Jundong Jiao

Abstract read
In one paragraph

Article in Nature communications, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Article
  6. Article
  7. Review
  8. Evaluating Animal Models for Improved Hemodialysis Vascular Access: A Focus on Arteriovenous Fistula.Medical science monitor : international medical journal of experimental and clinical research · 2025
    Review
  9. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Zhaozheng Li *Department of Nephrology, The Second Affiliated Hospital of Harbin Medical University, 150086, Harbin, China.ORCID http://orcid.org/0000-0003-4848-0798
Yao Zhao *Department of Nephrology, The Second Affiliated Hospital of Harbin Medical University, 150086, Harbin, China.
Zhenwei PanDepartment of Pharmacy at The Second Affiliated Hospital, Harbin Medical University, 150086, Harbin, China.ORCID http://orcid.org/0000-0002-1011-0954
Benzhi CaiDepartment of Pharmacy at The Second Affiliated Hospital, Harbin Medical University, 150086, Harbin, China.ORCID http://orcid.org/0000-0001-7590-6411
Chengwei ZhangDepartment of Nephrology, The Second Affiliated Hospital of Harbin Medical University, 150086, Harbin, China. zhangchengwei1981@163.com.ORCID http://orcid.org/0000-0002-2110-5501
Jundong JiaoDepartment of Nephrology, The Second Affiliated Hospital of Harbin Medical University, 150086, Harbin, China. jiaojundong@163.com.

Funding

Heilongjiang Provincial Science and Technology Department (Science and Technology Department of Heilongjiang Province) GY2019YF0165National Natural Science Foundation of China (National Science Foundation of China) 82170756
6 · The paper itself

Abstract

Arteriovenous fistulas (AVFs) are the most common vascular access points for hemodialysis (HD), but they have a high incidence of postoperative dysfunction, mainly due to excessive neointimal hyperplasia (NIH). Our previous studies have revealed a highly conserved LncRNA-LncDACH1 as an important regulator of cardiomyocyte and fibroblast proliferation. Herein, we find that LncDACH1 regulates NIH in AVF in male mice with conditional knockout of smooth muscle cell-specific LncDACH1 and in male mice model of AVF with LncDACH1 overexpression by adeno-associated virus. Mechanistically, silence of LncDACH1 activates p-AKT through promoting the expression of heat shock protein 90 (HSP90) and serine/arginine-rich splicing factor protein kinase 1 (SRPK1). Moreover, LncDACH1 is transcriptionally activated by transcription factor KLF9 that binds directly to the promoter region of the LncDACH1 gene. In this work, during AVF NIH, LncDACH1 is downregulated by KLF9 and promotes NIH through the HSP90/ SRPK1/ AKT signaling axis.

Indexed as

HSP90 Heat-Shock ProteinsHyperplasiaKruppel-Like Transcription FactorsMyocytes, Smooth MuscleNeointimaProto-Oncogene Proteins c-aktRNA, Long NoncodingAnimalsArteriovenous FistulaCell ProliferationEye ProteinsHumansMaleMiceMice, Inbred C57BLMice, KnockoutDach1 protein, mouseEye ProteinsHSP90 Heat-Shock ProteinsKruppel-Like Transcription FactorsProtein Serine-Threonine KinasesProto-Oncogene Proteins c-aktRNA, Long Noncoding

Identifiers

PMID38702316
PMCPMC11068796

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.