Evidence mapPaperPMID 38728626Full record

ArticleAdvanced science (Weinheim, Baden-Wurttemberg, Germany)2024

Cyclo His-Pro Attenuates Muscle Degeneration in Murine Myopathy Models.

Alessia De Masi, Nadège Zanou, Keno Strotjohann, Dohyun Lee, Tanes I Lima, Xiaoxu Li, Jongsu Jeon, Nicolas Place, Hoe-Yune Jung, Johan Auwerx

Abstract read
In one paragraph

Article in Advanced science (Weinheim, Baden-Wurttemberg, Germany), 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Review
  2. Article
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  5. Article
  6. Cyclo His-Pro Attenuates Muscle Degeneration in Murine Myopathy Models.Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2024
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Alessia De MasiLaboratory of Integrative Systems Physiology, Institute of Bioengineering, École Polytechnique Fédérale de Lausanne, Lausanne, 1015, Switzerland.ORCID 0000-0001-8453-6790
Nadège ZanouInstitute of Sport Sciences and Department of Biomedical Sciences, Faculty of Biology-Medicine, University of Lausanne, Lausanne, 1015, Switzerland.
Keno StrotjohannLaboratory of Integrative Systems Physiology, Institute of Bioengineering, École Polytechnique Fédérale de Lausanne, Lausanne, 1015, Switzerland.
Dohyun LeeR&D Center, NovMetaPharma Co., Ltd, Pohang, 37668, South Korea.
Tanes I LimaLaboratory of Integrative Systems Physiology, Institute of Bioengineering, École Polytechnique Fédérale de Lausanne, Lausanne, 1015, Switzerland.
Xiaoxu LiLaboratory of Integrative Systems Physiology, Institute of Bioengineering, École Polytechnique Fédérale de Lausanne, Lausanne, 1015, Switzerland.
Jongsu JeonR&D Center, NovMetaPharma Co., Ltd, Pohang, 37668, South Korea.
Nicolas PlaceInstitute of Sport Sciences and Department of Biomedical Sciences, Faculty of Biology-Medicine, University of Lausanne, Lausanne, 1015, Switzerland.
Hoe-Yune JungR&D Center, NovMetaPharma Co., Ltd, Pohang, 37668, South Korea.
Johan AuwerxLaboratory of Integrative Systems Physiology, Institute of Bioengineering, École Polytechnique Fédérale de Lausanne, Lausanne, 1015, Switzerland.ORCID 0000-0002-5065-5393

Funding

École Polytechnique Fédérale de LausanneEuropean Research Council ERC-AdG-787702H2020 Marie Skłodowska-Curie Actions Healthage - 812830National Research Foundation of Korea NRF 2017K1A1A2013124NovMetaPharmaSwiss National Science Foundation SNSF 31003A_179435University of Lausanne
6 · The paper itself

Abstract

Among the inherited myopathies, a group of muscular disorders characterized by structural and metabolic impairments in skeletal muscle, Duchenne muscular dystrophy (DMD) stands out for its devastating progression. DMD pathogenesis is driven by the progressive degeneration of muscle fibers, resulting in inflammation and fibrosis that ultimately affect the overall muscle biomechanics. At the opposite end of the spectrum of muscle diseases, age-related sarcopenia is a common condition that affects an increasing proportion of the elderly. Although characterized by different pathological mechanisms, DMD and sarcopenia share the development of progressive muscle weakness and tissue inflammation. Here, the therapeutic effects of Cyclo Histidine-Proline (CHP) against DMD and sarcopenia are evaluated. In the mdx mouse model of DMD, it is shown that CHP restored muscle contractility and force production, accompanied by the reduction of fibrosis and inflammation in skeletal muscle. CHP furthermore prevented the development of cardiomyopathy and fibrosis in the diaphragm, the two leading causes of death for DMD patients. CHP also attenuated muscle atrophy and functional deterioration in a mouse model of age-related sarcopenia. These findings from two different models of muscle dysfunction hence warrant further investigation into the effects of CHP on muscle pathologies in animal models and eventually in patients.

Indexed as

Disease Models, AnimalMice, Inbred mdxMuscle, SkeletalMuscular Dystrophy, DuchenneSarcopeniaAnimalsMaleMiceMice, Inbred C57BLcardiomyopathyDuchenne muscular dystrophymitochondrial dysfunctionmuscle fibrosissarcopenia

Identifiers

PMID38728626
PMCPMC11267275

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.