Evidence mapPaperPMID 38745238Full record

ArticleStem cell research & therapy2024

Macrophages modulate fibrosis during newt lens regeneration.

Georgios Tsissios, Anthony Sallese, J Raul Perez-Estrada, Jared A Tangeman, Weihao Chen, Byran Smucker, Sophia C Ratvasky, Erika Grajales-Esquivel, Arielle Martinez, Kimberly J Visser and 5 more

Abstract read
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Article in Stem cell research & therapy, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

15 authors.

Georgios Tsissios *Department of Biology, Miami University, Oxford, OH, USA.
Anthony Sallese *Department of Biology, Miami University, Oxford, OH, USA.
J Raul Perez-EstradaDepartment of Biology, Miami University, Oxford, OH, USA.
Jared A TangemanDepartment of Biology, Miami University, Oxford, OH, USA.
Weihao ChenCenter for Visual Sciences at, Miami University, Oxford, OH, USA.
Byran SmuckerCenter for Visual Sciences at, Miami University, Oxford, OH, USA.
Sophia C RatvaskyDepartment of Biology, Miami University, Oxford, OH, USA.
Erika Grajales-EsquivelDepartment of Biology, Miami University, Oxford, OH, USA.ORCID 0000-0001-8423-416X
Arielle MartinezDepartment of Biology, Miami University, Oxford, OH, USA.
Kimberly J VisserCRTD/ Center for Regenerative Therapies Dresden, Technische Universität Dresden, Dresden, Germany.
Alberto Joven ArausDepartment of Cell and Molecular Biology, Karolinska Institute, Stockholm, Sweden.ORCID 0000-0002-0926-4665
Hui WangCenter for Visual Sciences at, Miami University, Oxford, OH, USA.
András SimonDepartment of Cell and Molecular Biology, Karolinska Institute, Stockholm, Sweden.ORCID 0000-0002-1018-1891
Maximina H YunCRTD/ Center for Regenerative Therapies Dresden, Technische Universität Dresden, Dresden, Germany.
Katia Del Rio-TsonisDepartment of Biology, Miami University, Oxford, OH, USA. delriok@miamioh.edu.ORCID 0000-0002-3051-6716

Funding

Deutsche Forschungsgemeinschaft 450807335Deutsche Forschungsgemeinschaft 497658823Deutsche Forschungsgemeinschaft DFG 22137416Karolinska Institutet Research Grant-Projektbidrag FS-2020-0007NEI NIH HHS R21 EY031865NEI NIH HHS R21 EY033916NEI NIH HHS RO1 EY027801NINDS NIH HHS F99 NS129167
6 · The paper itself

Abstract

backgroundPrevious studies have suggested that macrophages are present during lens regeneration in newts, but their role in the process is yet to be elucidated.

methodsHere we generated a transgenic reporter line using the newt, Pleurodeles waltl, that traces macrophages during lens regeneration. Furthermore, we assessed early changes in gene expression during lens regeneration using two newt species, Notophthalmus viridescens and Pleurodeles waltl. Finally, we used clodronate liposomes to deplete macrophages during lens regeneration in both species and tested the effect of a subsequent secondary injury after macrophage recovery.

resultsMacrophage depletion abrogated lens regeneration, induced the formation of scar-like tissue, led to inflammation, decreased iris pigment epithelial cell (iPEC) proliferation, and increased rates of apoptosis in the eye. Some of these phenotypes persisted throughout the last observation period of 100 days and could be attenuated by exogenous FGF2 administration. A distinct transcript profile encoding acute inflammatory effectors was established for the dorsal iris. Reinjury of the newt eye alleviated the effects of macrophage depletion, including the resolution of scar-like tissue, and re-initiated the regeneration process.

conclusionsTogether, our findings highlight the importance of macrophages for facilitating a pro-regenerative environment in the newt eye by regulating fibrotic responses, modulating the overall inflammatory landscape, and maintaining the proper balance of early proliferation and late apoptosis of the iPECs.

Indexed as

FibrosisLens, CrystallineMacrophagesRegenerationSalamandridaeAnimalsApoptosisCell ProliferationInflammationLens regenerationMacrophageTissue repair

Identifiers

PMID38745238
PMCPMC11094960

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.