Evidence mapPaperPMID 38750140Full record

ArticleScientific reports2024

Vinpocetine alleviated alveolar epithelial cells injury in experimental pulmonary fibrosis by targeting PPAR-γ/NLRP3/NF-κB and TGF-β1/Smad2/3 pathways.

Zeena A Hussein, Ahmed R Abu-Raghif, Nibras J Tahseen, Khalid A Rashed, Nada S Shaker, Hayder Adnan Fawzi

Abstract read
In one paragraph

Article in Scientific reports, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed.

  1. Article
  2. Antifibrotic Effects ofInternational journal of molecular sciences · 2026
    Article
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  4. Article
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  6. Article
  7. Article
  8. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Zeena A HusseinDepartment of Pharmacology, College of Medicine, Al-Nahrain University, Baghdad, Iraq.
Ahmed R Abu-RaghifDepartment of Pharmacology, College of Medicine, Al-Nahrain University, Baghdad, Iraq.
Nibras J TahseenDepartment of Pharmacology and Toxicology, College of Pharmacy, Al-Bayan University, Baghdad, Iraq.
Khalid A RashedYeovil District Hospital, Somerset, UK.
Nada S ShakerDepartment of Pharmacology and Toxicology, College of Pharmacy, Mustansiriyah University, Baghdad, Iraq.
Hayder Adnan FawziDepartment of Pharmacy, Al-Mustafa University College, Baghdad, Iraq. hayder.adnan2010@gmail.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

This study aimed to investigate the potential anti-fibrotic activity of vinpocetine in an experimental model of pulmonary fibrosis by bleomycin and in the MRC-5 cell line. Pulmonary fibrosis was induced in BALB/c mice by oropharyngeal aspiration of a single dose of bleomycin (5 mg/kg). The remaining induced animals received a daily dose of pirfenidone (as a standard anti-fibrotic drug) (300 mg/kg/PO) and vinpocetine (20 mg/kg/PO) on day 7 of the induction till the end of the experiment (day 21). The results of the experiment revealed that vinpocetine managed to alleviate the fibrotic endpoints by statistically improving (P ≤ 0.05) the weight index, histopathological score, reduced expression of fibrotic-related proteins in immune-stained lung sections, as well as fibrotic markers measured in serum samples. It also alleviated tissue levels of oxidative stress and inflammatory and pro-fibrotic mediators significantly elevated in bleomycin-only induced animals (P ≤ 0.05). Vinpocetine managed to express a remarkable attenuating effect in pulmonary fibrosis both in vivo and in vitro either directly by interfering with the classical TGF-β1/Smad2/3 signaling pathway or indirectly by upregulating the expression of Nrf2 enhancing the antioxidant system, activating PPAR-γ and downregulating the NLRP3/NF-κB pathway making it a candidate for further clinical investigation in cases of pulmonary fibrosis.

Indexed as

Alveolar Epithelial CellsPulmonary FibrosisSignal TransductionVinca AlkaloidsAnimalsBleomycinCell LineDisease Models, AnimalHumansMaleMiceMice, Inbred BALB CNF-kappa BNLR Family, Pyrin Domain-Containing 3 ProteinOxidative StressPPAR gammaBleomycinNF-kappa BNLR Family, Pyrin Domain-Containing 3 ProteinPPAR gammaSmad2 ProteinSmad3 ProteinTransforming Growth Factor beta1Vinca AlkaloidsvinpocetineBleomycin-induced pulmonary fibrosisMRC-5 cell lineNLRP3 inflammasomePPAR-γTGF-β1/Smad2/3 pathwayVinpocetine

Identifiers

PMID38750140
PMCPMC11096407

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.