Evidence map›Paper›PMID 38784118›Full record

ArticleFrontiers in physiology2024

Dietary fatty acids modulate oxidative stress response to air pollution but not to infection.

Ann-Kathrin Ziegler, Johan Kjellberg Jensen, Lucía Jiménez-Gallardo, Jenny Rissler, Anders Gudmundsson, Jan-Åke Nilsson, Caroline Isaksson

Abstract read
In one paragraph

Article in Frontiers in physiology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. The Impairment of Body Condition Transiently Increases Oxidative Stress: A Dietary Restriction Experiment in Partridges.Journal of experimental zoology. Part A, Ecological and integrative physiology · 2025
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Ann-Kathrin Ziegler *Department of Biology, Lund University, Lund, Sweden.
Johan Kjellberg Jensen *Department of Biology, Lund University, Lund, Sweden.
Lucía Jiménez-GallardoDepartment of Biodiversity, Ecology and Evolution, Complutense University of Madrid, Madrid, Spain.
Jenny RisslerErgonomics and Aerosol Technology, Department of Design Sciences, Lund University, Lund, Sweden.
Anders GudmundssonErgonomics and Aerosol Technology, Department of Design Sciences, Lund University, Lund, Sweden.
Jan-Åke NilssonDepartment of Biology, Lund University, Lund, Sweden.
Caroline IsakssonDepartment of Biology, Lund University, Lund, Sweden.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Anthropogenic changes to the environment expose wildlife to many pollutants. Among these, tropospheric ozone is of global concern and a highly potent pro-oxidant. In addition, human activities include several other implications for wildlife, e.g., changed food availability and changed distribution of pathogens in cities. These co-occurring habitat changes may interact, thereby modulating the physiological responses and costs related to anthropogenic change. For instance, many food items associated with humans (e.g., food waste and feeders for wild birds) contain relatively more ω6-than ω3-polyunsaturated fatty acids (PUFAs). Metabolites derived from ω6-PUFAs can enhance inflammation and oxidative stress towards a stimulus, whereas the opposite response is linked to ω3-derived metabolites. Hence, we hypothesized that differential intake of ω6-and ω3-PUFAs modulates the oxidative stress state of birds and thereby affects the responses towards pro-oxidants. To test this, we manipulated dietary ω6:ω3 ratios and ozone levels in a full-factorial experiment using captive zebra finches (

Indexed as

antioxidantsavianimmune functionmultiple stressorsnutritiontropospheric ozone

Identifiers

PMID38784118
PMCPMC11112072

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.