Evidence mapPaperPMID 38814895Full record

ArticlePloS one2024

The short-chain fatty acid propionate prevents ox-LDL-induced coronary microvascular dysfunction by alleviating endoplasmic reticulum stress in HCMECs.

Dan Hong, Wen Tang, Fei Li, Yating Liu, Xiao Fu, Qin Xu

Abstract read
In one paragraph

Article in PloS one, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Dan HongDepartment of Geriatric Medicine, Xiangya Hospital, Central South University, Hunan, China.ORCID https://orcid.org/0009-0005-3983-0989
Wen TangDepartment of Geriatric Medicine, Xiangya Hospital, Central South University, Hunan, China.
Fei LiDepartment of Geriatric Medicine, Xiangya Hospital, Central South University, Hunan, China.
Yating LiuDepartment of Geriatric Medicine, Xiangya Hospital, Central South University, Hunan, China.
Xiao FuDepartment of Hematology Medicine, Xiangya Hospital, Central South University, Hunan, China.
Qin XuDepartment of Cardiology Medicine, Brain Hospital of Hunan Province, Hunan, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Coronary microvascular dysfunction (CMD) is a critical pathogenesis of cardiovascular diseases. Lower endothelial nitric oxide synthase (eNOS) phosphorylation leads to reduced endothelium-derived relaxing factor nitric oxide (NO) generation, causing and accelerating CMD. Endoplasmic reticulum stress (ER stress) has been shown to reduce NO production in umbilical vein endothelial cells. Oxidized low-density lipoprotein (ox-LDL) damages endothelial cell function. However, the relationship between ox-LDL and coronary microcirculation has yet to be assessed. Short-chain fatty acid (SCFA), a fermentation product of the gut microbiome, could improve endothelial-dependent vasodilation in human adipose arterioles, but the effect of SCFA on coronary microcirculation is unclear. In this study, we found ox-LDL stimulated expression of ER chaperone GRP78. Further, we activated downstream PERK/eIF2a, IRE1/JNK, and ATF6 signaling pathways, decreasing eNOS phosphorylation and NO production in human cardiac microvascular endothelial. Furthermore, SCFA-propionate can inhibit ox-LDL-induced eNOS phosphorylation reduction and raise NO production; the mechanism is related to the inhibition of ER stress and downstream signaling pathways PERK/eIF2a, IRE1/JNK, and ATF6. In summary, we demonstrate that ox-LDL induced CMD by activating ER stress, propionate can effectively counteract the adverse effects of ox-LDL and protect coronary microcirculation function via inhibiting ER stress.

Indexed as

Endoplasmic Reticulum Chaperone BiPEndoplasmic Reticulum StressLipoproteins, LDLNitric OxideNitric Oxide Synthase Type IIIPropionatesSignal TransductionActivating Transcription Factor 6Coronary VesselseIF-2 KinaseEndothelial CellsFatty Acids, VolatileHeat-Shock ProteinsHumansMicrocirculationPhosphorylationActivating Transcription Factor 6eIF-2 KinaseEndoplasmic Reticulum Chaperone BiPFatty Acids, VolatileHeat-Shock ProteinsHSPA5 protein, humanLipoproteins, LDLNitric OxideNitric Oxide Synthase Type IIINOS3 protein, humanoxidized low density lipoproteinPropionates

Identifiers

PMID38814895
PMCPMC11139260

What Socratic holds

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LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.