Evidence mapPaperPMID 38884652Full record

ArticlePsychopharmacology2025

Effect of the glucagon-like peptide-1 (GLP-1) receptor agonist semaglutide on alcohol consumption in alcohol-preferring male vervet monkeys.

Anders Fink-Jensen, Gitta Wörtwein, Mette Kruse Klausen, Jens Juul Holst, Bolette Hartmann, Morgan Thomsen, Maurice Ptito, Amy Beierschmitt, Roberta M Palmour

Abstract read
In one paragraph

Article in Psychopharmacology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 27 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
27citing papers in PubMed, 2 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

27 citing papers in PubMed, 2 syntheses or guidelines pooled it.

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  17. Non-invasive tests for MetALD and alcohol-related liver disease.JHEP reports : innovation in hepatology · 2025
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Anders Fink-JensenLaboratory of Neuropsychiatry, Mental Health Centre Copenhagen, Mental Health Services in the Capital Region of Denmark, University of Copenhagen, Hovedvejen 17, Frederiksberg, DK-2000, Denmark. anders.fink-jensen@regionh.dk.
Gitta WörtweinLaboratory of Neuropsychiatry, Mental Health Centre Copenhagen, Mental Health Services in the Capital Region of Denmark, University of Copenhagen, Hovedvejen 17, Frederiksberg, DK-2000, Denmark.
Mette Kruse KlausenLaboratory of Neuropsychiatry, Mental Health Centre Copenhagen, Mental Health Services in the Capital Region of Denmark, University of Copenhagen, Hovedvejen 17, Frederiksberg, DK-2000, Denmark.
Jens Juul HolstNovo Nordisk Foundation Center for Basic Metabolic Research, Department of Biomedical Sciences, University of Copenhagen, Copenhagen, Denmark.
Bolette HartmannNovo Nordisk Foundation Center for Basic Metabolic Research, Department of Biomedical Sciences, University of Copenhagen, Copenhagen, Denmark.
Morgan ThomsenLaboratory of Neuropsychiatry, Mental Health Centre Copenhagen, Mental Health Services in the Capital Region of Denmark, University of Copenhagen, Hovedvejen 17, Frederiksberg, DK-2000, Denmark.
Maurice PtitoDepartment of Neuroscience, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark.
Amy BeierschmittBehavioural Science Foundation, St. Kitts, Saint Kitts and Nevis.
Roberta M PalmourBehavioural Science Foundation, St. Kitts, Saint Kitts and Nevis.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

rationaleGlucagon-like peptide-1 (GLP-1) receptor agonists reduce alcohol consumption in rodents and non-human primates. Semaglutide is a new long-acting GLP-1 receptor agonist, widely used in the clinic against type 2 diabetes and obesity. It is also reported to reduce alcohol intake in rodents.

objectivesThis study investigates the possible inhibitory effect of semaglutide on alcohol intake in alcohol-preferring African green monkeys.

methodsWe performed a vehicle-controlled study on male monkeys that had demonstrated a preference for alcohol. In the monkeys selected for voluntary alcohol drinking, alcohol consumption was measured for ten days at baseline (Monday to Friday for two weeks). During this period, the monkeys had access to alcohol 4 h per day and free access to water 24 h per day. After two weeks of baseline measurements, the monkeys were randomized to semaglutide or vehicle. Each group consisted of ten monkeys, and the two groups were balanced with respect to baseline alcohol intake. Following the baseline period, the monkeys were treated with escalating doses of semaglutide (up to 0.05 mg/kg) or vehicle subcutaneously twice weekly for two weeks during which period alcohol was not available. After uptitration, the monkeys had access to alcohol 4 h daily for 20 days (Monday to Friday for 4 weeks), and alcohol consumption was measured. During this alcohol exposure period, treatment with semaglutide (0.05 mg/kg twice weekly) or vehicle continued for three weeks followed by a one-week washout period.

resultsCompared to the vehicle, semaglutide significantly reduced alcohol intake. There were no signs of emetic events or changes in water intake.

conclusionsThese data demonstrate for the first time the potent effect of semaglutide in reducing voluntary alcohol intake in non-human primates and further substantiate the need for clinical trials investigating the effect of semaglutide in patients with alcohol-use disorder.

Indexed as

Alcohol DrinkingGlucagon-Like Peptide-1 Receptor AgonistsGlucagon-Like PeptidesAnimalsChlorocebus aethiopsEthanolGlucagon-Like Peptide 1Glucagon-Like Peptide-1 ReceptorMaleSemaglutideEthanolGlucagon-Like Peptide 1Glucagon-Like Peptide-1 ReceptorGlucagon-Like Peptide-1 Receptor AgonistsGlucagon-Like PeptidesSemaglutideAlcoholAlcohol use disorderAUDGLP-1MonkeyNon-human primatesSelf administrationSemaglutideVervet

Identifiers

PMID38884652
PMCPMC11742737

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.