Evidence map›Paper›PMID 38892255›Full record

ArticleInternational journal of molecular sciences2024

Metabolic Profile and Lipid Metabolism Phenotype in Mice with Conditional Deletion of Hepatic BMAL1.

Weijia Gu, Ting Li, Yuxin Huang, Ruiqing Wang, Lu Zhang, Rucheng Chen, Ran Li, Cuiqing Liu

Abstract read
In one paragraph

Article in International journal of molecular sciences, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed.

  1. Article
  2. Review
  3. Article
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  8. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Weijia GuSchool of Public Health, Zhejiang Chinese Medical University, Hangzhou 310053, China.ORCID 0000-0003-2613-0981
Ting LiSchool of Public Health, Zhejiang Chinese Medical University, Hangzhou 310053, China.
Yuxin HuangSchool of Public Health, Zhejiang Chinese Medical University, Hangzhou 310053, China.
Ruiqing WangSchool of Public Health, Zhejiang Chinese Medical University, Hangzhou 310053, China.
Lu ZhangSchool of Public Health, Zhejiang Chinese Medical University, Hangzhou 310053, China.
Rucheng ChenSchool of Public Health, Zhejiang Chinese Medical University, Hangzhou 310053, China.
Ran LiSchool of Public Health, Zhejiang Chinese Medical University, Hangzhou 310053, China.ORCID 0000-0002-6268-8811
Cuiqing LiuSchool of Public Health, Zhejiang Chinese Medical University, Hangzhou 310053, China.

Funding

2023 College Student Innovation and Entrepreneurship Training Program 202310344030Medical Health Science and Technology Project of Zhejiang Provincial Health Commission 2019KY471Medical Health Science and Technology Project of Zhejiang Provincial Health Commission 2019KY472National Natural Science Foundation of China 81904027National Natural Science Foundation of China 81973001National Natural Science Foundation of China 82273590Zhejiang Provincial College Student Science and Technology Innovation Projects 2023R410030
6 · The paper itself

Abstract

The disruption of circadian rhythms (CRs) has been linked to metabolic disorders, yet the role of hepatic BMAL1, a key circadian regulator, in the whole-body metabolism and the associated lipid metabolic phenotype in the liver remains unclear.

Indexed as

ARNTL Transcription FactorsCircadian RhythmHepatocytesLipid MetabolismLiverMice, Inbred C57BLMice, KnockoutAnimalsGene DeletionLipogenesisMaleMetabolomeMicePhenotypeARNTL Transcription FactorsBmal1 protein, mouseBMAL1circadian rhythmlipid metabolismmitochondrial β-oxidationtriglyceride

Identifiers

PMID38892255
PMCPMC11172555

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.