Evidence mapPaperPMID 3891471Full record

ArticleDiabetes1985

The disposal of an oral glucose load in healthy subjects. A quantitative study.

E Ferrannini, O Bjorkman, G A Reichard, A Pilo, M Olsson, J Wahren, R A DeFronzo

2 registry-linked trialsAbstract read
PubMed Publisher
In one paragraph

Article in Diabetes, 1985. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to 2 registered trials, which are not on this map. Cited by 118 papers, 3 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
118citing papers in PubMed, 3 pooled it
8.9field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT02613897 nacompletedstarted 2016, after this paper: background citation

A 16-wk, Uni-center, Randomized, Double-blind, Parallel, Phase 3b Trial to Evaluate Efficacy of Saxagliptin + Dapagliflozin vs.Dapagliflozin With Regard to EGP in T2DM With Insufficient Glycemic Control on Metformin+/-Sulfonylurea Therapy

Ran2016Enrolled56Registered outcomes9Posted comparisons0ConditionsDiabetes Mellitus, Type 2Armsdapagliflozin, Placebo, Saxagliptin
Open the trial in the graph
NCT07690176 narecruitingnot on this mapstarted 2026, after this paper: background citation

The Metabolic Effects of a Hyperglycaemic Meal in Lean and Obese Individuals Using a [14C]-Glucose Microtracer Approach

TypeinterventionalSponsorWageningen UniversityRan2026 to 2026Enrolled24ConditionsObesity & OverweightArmsLow-glycemic breakfast (randomized vs high glycemic), High-glycaemic breakfast (randomized vs low glycemic), High-glycaemic breakfast
3 · Its place in the literature

Who cites it

118 citing papers in PubMed, 3 syntheses or guidelines pooled it, 418 citations in OpenAlex.

  1. Pooled it
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  3. Effect of blood glucose level on standardized uptake value (SUV) inEuropean journal of nuclear medicine and molecular imaging · 2019
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58 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 2 countries.

E Ferrannini
O Bjorkman
G A Reichard
A Pilo
M Olsson
J Wahren
R A DeFronzo
University of Pisa · ITKarolinska Institutet · SE

Funding

GLUCOREGULATORY HORMONE INTERACTIONS IN DIABETESR01AM020495 · YALE UNIVERSITY · 1985 to 1985
REGULATION OF HEPATIC AND PERIPHERAL GLUCOSE METABOLISMR01AM024092 · YALE UNIVERSITY · 1985 to 1985
NCRR NIH HHS RR-125NIADDK NIH HHS AM-20495NIADDK NIH HHS AM-24092
6 · The paper itself

Abstract

Although it is an established concept that the liver is important in the disposition of glucose, the quantitative contribution of the splanchnic and peripheral tissues, respectively, to the disposal of an oral glucose load is still controversial. In the present investigation, we have employed the hepatic venous catheter technique in combination with a double-tracer approach (in which the glucose pool is labeled with 3H-glucose and the oral glucose load is labeled with 14C-glucose) to quantitate the four determinants of oral glucose tolerance: rate of oral glucose appearance, splanchnic glucose uptake, peripheral glucose uptake, and suppression of hepatic glucose production. Studies were carried out in 11 normal volunteers in the overnight-fasted state and for 3.5 h after the ingestion of glucose (1 g/kg body wt; range, 55-93 g). In the postabsorptive state, the rate of endogenous (hepatic) glucose production, evaluated from the 3H-glucose infusion, was 2.34 +/- 0.06 mg/min X kg. Glucose ingestion was accompanied by a prompt reduction of endogenous glucose output, which reached a nadir of 0.62 +/- 0.23 mg/min X kg at 45 min and remained suppressed after 3.5 h (0.85 +/- 0.22 mg/min X kg). The average inhibition of hepatic glucose output during the absorptive period was 53 +/- 5%. The appearance of ingested glucose in arterial blood, as derived from the 14C-glucose measurements after correction for recycling 14-C radioactivity, reached a peak after 15-30 min, and 14C-glucose continued to enter the systemic circulation throughout the observation period. The rate of appearance of ingested glucose was 2.47 +/- 0.45 mg/min X kg at 3.5 h. A total of 73 +/- 4% of the oral load was recovered in the systemic circulation within 3.5 h.(ABSTRACT TRUNCATED AT 250 WORDS)

Indexed as

Glucose Tolerance TestAdministration, OralAdultBlood GlucoseFemaleGlucoseHumansInsulinMaleMiddle AgedSplanchnic CirculationTime FactorsBlood GlucoseGlucoseInsulin

Identifiers

PMID3891471
OpenAlexW2003719947

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.