Evidence mapPaperPMID 38965243Full record

ReviewSignal transduction and targeted therapy2024

Cancer stem cells: advances in knowledge and implications for cancer therapy.

Xianjing Chu, Wentao Tian, Jiaoyang Ning, Gang Xiao, Yunqi Zhou, Ziqi Wang, Zhuofan Zhai, Guilong Tanzhu, Jie Yang, Rongrong Zhou

Abstract readReview
In one paragraph

Review in Signal transduction and targeted therapy, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 322 papers, 3 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
322citing papers in PubMed, 3 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

322 citing papers in PubMed, 3 syntheses or guidelines pooled it.

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  18. Modulation ofInternational journal of molecular sciences · 2026
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262 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Xianjing Chu *Department of Oncology, Xiangya Hospital, Central South University, Changsha, 410008, China.
Wentao Tian *Department of Oncology, Xiangya Hospital, Central South University, Changsha, 410008, China.
Jiaoyang Ning *Department of Oncology, Xiangya Hospital, Central South University, Changsha, 410008, China.
Gang XiaoDepartment of Oncology, Xiangya Hospital, Central South University, Changsha, 410008, China.
Yunqi ZhouDepartment of Oncology, Xiangya Hospital, Central South University, Changsha, 410008, China.
Ziqi WangDepartment of Oncology, Xiangya Hospital, Central South University, Changsha, 410008, China.
Zhuofan ZhaiDepartment of Oncology, Xiangya Hospital, Central South University, Changsha, 410008, China.
Guilong TanzhuDepartment of Oncology, Xiangya Hospital, Central South University, Changsha, 410008, China. tzgl2716@126.com.
Jie YangDepartment of Oncology, Xiangya Hospital, Central South University, Changsha, 410008, China. yangjie@csu.edu.cn.
Rongrong ZhouDepartment of Oncology, Xiangya Hospital, Central South University, Changsha, 410008, China. zhourr@csu.edu.cn.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cancer stem cells (CSCs), a small subset of cells in tumors that are characterized by self-renewal and continuous proliferation, lead to tumorigenesis, metastasis, and maintain tumor heterogeneity. Cancer continues to be a significant global disease burden. In the past, surgery, radiotherapy, and chemotherapy were the main cancer treatments. The technology of cancer treatments continues to develop and advance, and the emergence of targeted therapy, and immunotherapy provides more options for patients to a certain extent. However, the limitations of efficacy and treatment resistance are still inevitable. Our review begins with a brief introduction of the historical discoveries, original hypotheses, and pathways that regulate CSCs, such as WNT/β-Catenin, hedgehog, Notch, NF-κB, JAK/STAT, TGF-β, PI3K/AKT, PPAR pathway, and their crosstalk. We focus on the role of CSCs in various therapeutic outcomes and resistance, including how the treatments affect the content of CSCs and the alteration of related molecules, CSCs-mediated therapeutic resistance, and the clinical value of targeting CSCs in patients with refractory, progressed or advanced tumors. In summary, CSCs affect therapeutic efficacy, and the treatment method of targeting CSCs is still difficult to determine. Clarifying regulatory mechanisms and targeting biomarkers of CSCs is currently the mainstream idea.

Indexed as

NeoplasmsNeoplastic Stem CellsDrug Resistance, NeoplasmHumansSignal Transduction

Identifiers

PMID38965243
PMCPMC11224386

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.