Evidence map›Paper›PMID 38999849›Full record

SynthesisNutrients2024

Intermittent Fasting Attenuates Obesity-Induced Triple-Negative Breast Cancer Progression by Disrupting Cell Cycle, Epithelial-Mesenchymal Transition, Immune Contexture, and Proinflammatory Signature.

Deok-Soo Son, Kaitlyn A Done, Jubin Son, Michael G Izban, Carlos Virgous, Eun-Sook Lee, Samuel E Adunyah

Abstract readMeta-Analysis
In one paragraph

Synthesis in Nutrients, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Trial
  2. Article
  3. Review
  4. Review
  5. Review
  6. Article
  7. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Deok-Soo SonDepartment of Biochemistry, Cancer Biology, Neuroscience and Pharmacology, School of Medicine, Meharry Medical College, Nashville, TN 37208, USA.
Kaitlyn A DoneBiochemistry Program, College of Arts and Sciences, Spelman College, Atlanta, GA 30314, USA.
Jubin SonNeuroscience Program, College of Arts and Sciences, The University of Tennessee, Knoxville, TN 37996, USA.
Michael G IzbanPathology, Anatomy and Cell Biology, Meharry Medical College, Nashville, TN 37208, USA.
Carlos VirgousAnimal Core Facility, Meharry Medical College, Nashville, TN 37208, USA.
Eun-Sook LeeDepartment of Pharmaceutical Sciences, College of Pharmacy, Florida A&M University, Tallahassee, FL 32301, USA.ORCID 0000-0001-7047-5288
Samuel E AdunyahDepartment of Biochemistry, Cancer Biology, Neuroscience and Pharmacology, School of Medicine, Meharry Medical College, Nashville, TN 37208, USA.

Funding

TISSUE CoreP50CA098131 · NCI · VANDERBILT UNIVERSITY MEDICAL CENTER · PI PARK, BEN HO, PIETENPOL, JENNIFER A · 2003 to 2024
$48.7M
The RCMI Program in Health Disparities Research at Meharry Medical College - SupplementU54MD007586 · NIMHD · MEHARRY MEDICAL COLLEGE · PI Samuel Evans Adunyah, JAMES E HILDRETH · 2017 to 2026
$48.2M
Translational Pathology CoreU54CA163069 · NCI · MEHARRY MEDICAL COLLEGE · PI ADUNYAH, SAMUEL EVANS · 2011 to 2025
$18.8M
Mechanisms associated with neuroprotection from Mn-induced neurotoxicity.R01ES031282 · NIEHS · FLORIDA AGRICULTURAL AND MECHANICAL UNIV · PI LEE, EUNSOOK YU · 2020 to 2025
$2.4M
Roles of proinflammatory chemokines linking obesity and breast cancerSC1CA200519 · NCI · MEHARRY MEDICAL COLLEGE · PI SON, DEOK-SOO · 2015 to 2018
$1.5M
ACS DICRIDG-21-071-01-DICRIDGNCI NIH HHS P50 CA098131NCI NIH HHS SC1 CA200519NCI NIH HHS U54 CA163069NIEHS NIH HHS R01 ES031282NIH HHS 1P50CA098131-21A1NIH HHS 1R01ES031282-23A1NIH HHS 1SC1CA200519-21A1NIH HHS 1U54CA163069-21A1NIH HHS 5U54MD007586-22A1NIMHD NIH HHS U54 MD007586
6 · The paper itself

Abstract

Obesity is associated with one-fifth of cancer deaths, and breast cancer is one of the obesity-related cancers. Triple-negative breast cancer (TNBC) lacks estrogen and progesterone receptors and human epidermal growth factor receptor 2, leading to the absence of these therapeutic targets, followed by poor overall survival. We investigated if obesity could hasten TNBC progression and intermittent fasting (IF) could attenuate the progression of obesity-related TNBC. Our meta-analysis of the TNBC outcomes literature showed that obesity led to poorer overall survival in TNBC patients. Fasting-mimicking media reduced cell proliferation disrupted the cell cycle, and decreased cell migration and invasion. IF decreased body weight in obese mice but no change in normal mice. Obese mice exhibited elevated plasma glucose and cholesterol levels, increased tumor volume and weight, and enhanced macrophage accumulation in tumors. The obesity-exacerbated TNBC progression was attenuated after IF, which decreased cyclin B1 and vimentin levels and reduced the proinflammatory signature in the obesity-associated tumor microenvironment. IF attenuated obesity-induced TNBC progression through reduced obesity and tumor burdens in cell and animal experiments, supporting the potential of a cost-effective adjuvant IF therapy for TNBC through lifestyle change. Further evidence is needed of these IF benefits in TNBC, including from human clinical trials.

Indexed as

Cell CycleDisease ProgressionEpithelial-Mesenchymal TransitionFastingObesityTriple Negative Breast NeoplasmsAnimalsCell Line, TumorCell MovementCell ProliferationFemaleHumansInflammationIntermittent FastingMiceMice, Obeseepithelial–mesenchymal transitionintermittent fastingobesityproinflammatory signaturetriple-negative breast cancer

Identifiers

PMID38999849
PMCPMC11243652

What Socratic holds

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LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.