Evidence mapPaperPMID 39015377Full record

ArticleFrontiers in neuroscience2024

Sustained corneal nerve loss predicts the development of diabetic neuropathy in type 2 diabetes.

Georgios Ponirakis, Ibrahim Al-Janahi, Einas Elgassim, Moayad Homssi, Ioannis N Petropoulos, Hoda Gad, Adnan Khan, Hadeel B Zaghloul, Hamda Ali, Mashhood A Siddique and 18 more

Abstract read
In one paragraph

Article in Frontiers in neuroscience, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

28 authors.

Georgios PonirakisDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Ibrahim Al-JanahiNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Einas ElgassimDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Moayad HomssiDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Ioannis N PetropoulosDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Hoda GadDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Adnan KhanDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Hadeel B ZaghloulDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Hamda AliNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Mashhood A SiddiqueNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Fatima F S MohamedDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Lina H M AhmedDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Youssra DakrouryDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Abeer M M El ShewehyDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Ruba SaeidDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Fadwa MahjoubDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Shaikha N Al-ThaniDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Farheen AhmedDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Rawan HusseinDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Salah MahmoudDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Nebras H HadidDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Aisha Al ObaidanDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Iuliia SalivonNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Ziyad R MahfoudDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Mahmoud A ZirieNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Yousuf Al-AnsariNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Stephen L AtkinRoyal College of Surgeons in Ireland Bahrain, Adliya, Bahrain.
Rayaz A MalikDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Introduction: This study was undertaken to investigate whether sustained rather than a single measure of corneal nerve loss was associated with the onset of diabetic peripheral neuropathy (DPN) and the progression of neuropathic symptoms and deficits in individuals with type 2 diabetes (T2D). Methods: Participants underwent clinical, metabolic testing and assessment of neuropathic symptoms, vibration perception threshold (VPT), sudomotor function, and corneal confocal microscopy (CCM) at baseline, 1, 2, and 4-7 years. Sustained corneal nerve loss was defined as abnormal corneal nerve fiber density (CNFD, <24 fibers/mm Results: A total of 107 participants with a mean duration of T2D of 13.3 ± 7.3 years, aged 54.8 ± 8.5 years, underwent baseline and follow-up assessments over a median duration of 4 years, ranging from 1 to 7 years. The DPN prevalence at baseline was 18/107 (16.8%), and of the 89 participants without DPN at baseline, 13 (14.6%) developed DPN during follow-up. Approximately half of the cohort had sustained corneal nerve damage, and corneal nerve measures were significantly lower in this group than those without sustained damage ( Conclusion: Sustained abnormal CCM is associated with more severe corneal nerve damage, DPN development, and the progression of neuropathic symptoms and deficits. Regular CCM monitoring may enable the identification of those at greater risk of developing and worsening DPN who may benefit from more aggressive risk factor reduction.

Indexed as

corneal confocal microscopydiabetic neuropathypredictive biomarkerrisk factor reductionsustained corneal nerve damagetype 2 diabetes

Identifiers

PMID39015377
PMCPMC11249546

What Socratic holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.