Evidence mapPaperPMID 39063997Full record

ArticleJournal of personalized medicine2024

The Role of Pro-Inflammatory Chemokines CCL-1, 2, 4, and 5 in the Etiopathogenesis of Type 2 Diabetes Mellitus in Subjects from the Asir Region of Saudi Arabia: Correlation with Different Degrees of Obesity.

Mohammad Muzaffar Mir, Jaber Alfaifi, Shahzada Khalid Sohail, Syeda Fatima Rizvi, Md Tanwir Akhtar, Mushabab Ayed Abdullah Alghamdi, Rashid Mir, Javed Iqbal Wani, Zia Ul Sabah, Fahad A Alhumaydhi and 6 more

Abstract read
In one paragraph

Article in Journal of personalized medicine, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

16 authors.

Mohammad Muzaffar MirDepartment of Basic Medical Sciences, College of Medicine, University of Bisha, Bisha 61922, Saudi Arabia.ORCID 0000-0003-2068-3075
Jaber AlfaifiDepartment of Child Health, College of Medicine, University of Bisha, Bisha 61922, Saudi Arabia.ORCID 0000-0002-4507-3970
Shahzada Khalid SohailDepartment of Basic Medical Sciences, College of Medicine, University of Bisha, Bisha 61922, Saudi Arabia.ORCID 0000-0002-7286-8074
Syeda Fatima RizviDepartment of Basic Medical Sciences, College of Medicine, University of Bisha, Bisha 61922, Saudi Arabia.ORCID 0009-0000-5883-7362
Md Tanwir AkhtarDepartment of Public Health, College of Health Sciences, Saudi Electronic University, Riyadh 93499, Saudi Arabia.ORCID 0000-0002-9412-3815
Mushabab Ayed Abdullah AlghamdiDepartment of Internal Medicine, College of Medicine, University of Bisha, Bisha 61922, Saudi Arabia.ORCID 0000-0002-8675-5871
Rashid MirPrince Fahd Bin Sultan Research Chair, Department of MLT, Faculty of Applied Medical Sciences, University of Tabuk, Tabuk 71491, Saudi Arabia.
Javed Iqbal WaniDepartment of Internal Medicine, College of Medicine, King Khalid University, Abha 61421, Saudi Arabia.ORCID 0000-0002-4605-6670
Zia Ul SabahDepartment of Internal Medicine, College of Medicine, King Khalid University, Abha 61421, Saudi Arabia.ORCID 0000-0003-4473-7373
Fahad A AlhumaydhiDepartment of Medical Laboratories, College of Applied Medical Sciences, Qassim University, Buraydah 51452, Saudi Arabia.ORCID 0000-0002-0151-8309
Fahad AlremthiDiabetes and Endocrine Center, King Abdullah Hospital, Ministry of Health, Bisha 61922, Saudi Arabia.ORCID 0000-0002-8393-6765
AbdulElah Al Jarallah AlQahtaniDepartment of Internal Medicine, College of Medicine, University of Bisha, Bisha 61922, Saudi Arabia.
Muffarah Hamid AlharthiDepartment of Family Medicine, College of Medicine, University of Bisha, Bisha 61922, Saudi Arabia.
Masoud Ishag Elkhalifa AdamDepartment of Medical Education, College of Medicine, University of Bisha, Bisha 61922, Saudi Arabia.
Imadeldin ElfakiDepartment of Biochemistry, Faculty of Science, University of Tabuk, Tabuk 71491, Saudi Arabia.
Hany M A SonpolDepartment of Basic Medical Sciences, College of Medicine, University of Bisha, Bisha 61922, Saudi Arabia.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundType 2 diabetes mellitus (T2DM) is becoming a major global health concern, especially in developing nations. The high prevalence of obesity and related diabetes cases are attributed to rapid economic progress, physical inactivity, the consumption of high-calorie foods, and changing lifestyles.

objectivesWe investigated the roles of pro-inflammatory chemokines CCL1, 2, 4, and 5 in T2DM with varying levels of obesity in the Asir region of Saudi Arabia. MATERIALS AND

methodsIn total, 170 confirmed T2DM subjects and a normal control group were enrolled. Demographic data, serum levels of CCL-1, 2, 4, and 5, and biochemical indices were assessed in the subjects and control groups by standard procedures.

resultsT2DM subjects were divided into four groups: A (normal body weight), B (overweight), C (obese), and D (highly obese). We observed that male and female control subjects had similar mean serum concentrations of pro-inflammatory chemokines CCL-1, 2, 4, and 5. T2DM subjects in all the four groups showed significantly higher levels of all the four chemokines compared to the controls, regardless of gender. In T2DM subjects with obesity and severe obesity, the rise was most significant. There was a progressive rise in the concentrations of CCL-1, 2, and 4 in T2DM subjects with increasing BMI. Serum CCL5 levels increased significantly in all T2DM subject groups. The increase in CCL5 was more predominant in normal-weight people, compared to overweight and obese T2DM subjects.

conclusionsMale and female control subjects had similar serum levels of pro-inflammatory chemokines CCL-1, 2, 4, and 5. The progressive rise in blood concentrations of three pro-inflammatory chemokines CCL-1, 2, and 4 in T2DM subjects with increasing BMI supports the idea that dyslipidemia and obesity contribute to chronic inflammation and insulin resistance. Serum CCL5 levels increased significantly in all T2DM subject groups. The selective and more pronounced increase in CCL5 in the T2DM group with normal BMI, compared to subjects with varying degrees of obesity, was rather surprising. Further research is needed to determine if CCL5 underexpression in overweight and obese T2DM subjects is due to some unexplained counterbalancing processes.

Indexed as

CCL1CCL2CCL4CCL5obesitypro-inflammatory chemokinestype 2 diabetes mellitus

Identifiers

PMID39063997
PMCPMC11277753

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.