Evidence map›Paper›PMID 39076267›Full record

ReviewReviews in cardiovascular medicine2023

New Concepts on the Pathophysiology of Acute Coronary Syndrome.

Deqiang Yuan, Jiapeng Chu, Jun Qian, Hao Lin, Guoqi Zhu, Fei Chen, Xuebo Liu

Abstract readReview
In one paragraph

Review in Reviews in cardiovascular medicine, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed.

  1. Trial
  2. Article
  3. Optical Coherence Tomography in Acute Coronary Syndromes.Reviews in cardiovascular medicine · 2025
    Review
  4. Review
  5. Review
  6. Review
  7. Article
  8. Review
  9. Article
  10. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Deqiang YuanDepartment of Cardiology, Tongji Hospital, School of Medicine, Tongji University, 200065 Shanghai, China.
Jiapeng ChuDepartment of Cardiology, Tongji Hospital, School of Medicine, Tongji University, 200065 Shanghai, China.
Jun QianDepartment of Cardiology, Tongji Hospital, School of Medicine, Tongji University, 200065 Shanghai, China.
Hao LinDepartment of Cardiology, Tongji Hospital, School of Medicine, Tongji University, 200065 Shanghai, China.
Guoqi ZhuDepartment of Cardiology, Tongji Hospital, School of Medicine, Tongji University, 200065 Shanghai, China.
Fei ChenDepartment of Cardiology, Tongji Hospital, School of Medicine, Tongji University, 200065 Shanghai, China.
Xuebo LiuDepartment of Cardiology, Tongji Hospital, School of Medicine, Tongji University, 200065 Shanghai, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Acute coronary syndrome (ACS) is the most severe form of ischemic heart disease. Although it is caused by atherosclerotic plaque thrombosis or nonatherosclerotic causes, its pathophysiological mechanism of ACS is not fully understood, and its concept is constantly updated and developed. At present, the main pathophysiological mechanisms include plaque rupture, plaque erosion, calcified nodules (CN) and non-atherosclerotic causes such as coronary vasospasm and myocardial bridging (MB). These mechanisms may overlap and coexist in some ACS patients. Therefore, the pathophysiological mechanism of ACS is complex, and is of great significance for the diagnosis and treatment of ACS. This review will discuss the pathophysiological mechanisms of ACS to provide new thoughts on the pathogenesis, diagnosis and treatment of ACS.

Indexed as

acute coronary syndromesatherosclerosiscalcified nodulesplaque erosionplaque rupture

Identifiers

PMID39076267
PMCPMC11273028

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.