Evidence map›Paper›PMID 39087468›Full record

ReviewThe Journal of clinical investigation2024

CHIP: a clonal odyssey of the bone marrow niche.

Wolfgang E Schleicher, Bridget Hoag, Marco De Dominici, James DeGregori, Eric M Pietras

Abstract readReview
In one paragraph

Review in The Journal of clinical investigation, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed.

  1. Review
  2. Review
  3. Review
  4. Translational Geroscience Strategies for Delaying Multimorbidity.ACS pharmacology & translational science · 2026
    Review
  5. Review
  6. Review
  7. Article
  8. Review
  9. Review
  10. Review
  11. The emergence of clonal hematopoiesis as a disease determinant.The Journal of clinical investigation · 2024
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Wolfgang E SchleicherDivision of Hematology, Department of Medicine, and.
Bridget HoagDepartment of Biochemistry and Molecular Genetics, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA.
Marco De DominiciDepartment of Biochemistry and Molecular Genetics, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA.
James DeGregoriDivision of Hematology, Department of Medicine, and.
Eric M PietrasDivision of Hematology, Department of Medicine, and.

Funding

Impact of IL-1 signaling on hematopoietic stem cell function and emergence of clonal hematopoiesis.R01DK119394 · NIDDK · UNIVERSITY OF COLORADO DENVER · PI PIETRAS, ERIC M · 2019 to 2023
$2.0M
Predoctoral Training in the Genetics of Development, Disease and RegenerationT32GM141742 · NIGMS · UNIVERSITY OF COLORADO DENVER · PI Bruce H Appel, Jeffrey Kyle Moore · 2021 to 2026
$1.9M
Aberrant glycolysis as a driver of mutant HSPC expansion in clonal hematopoiesisR01DK137183 · NIDDK · UNIVERSITY OF COLORADO DENVER · PI Eric M Pietras · 2023 to 2026
$1.8M
Determining how aging-associated changes in the microenvironment contribute to leukemogenesisR01AG067584 · NIA · UNIVERSITY OF COLORADO DENVER · PI DEGREGORI, JAMES V · 2019 to 2022
$1.7M
Aged tissue environments as drivers of oncogenic adaptation in hematopoiesisR01AG066544 · NIA · UNIVERSITY OF COLORADO DENVER · PI DEGREGORI, JAMES V · 2020 to 2024
$1.1M
NIA NIH HHS R01 AG066544NIA NIH HHS R01 AG067584NIDDK NIH HHS R01 DK119394NIDDK NIH HHS R01 DK137183NIGMS NIH HHS T32 GM141742
6 · The paper itself

Abstract

Clonal hematopoiesis of indeterminate potential (CHIP) is characterized by the selective expansion of hematopoietic stem and progenitor cells (HSPCs) carrying somatic mutations. While CHIP is typically asymptomatic, it has garnered substantial attention due to its association with the pathogenesis of multiple disease conditions, including cardiovascular disease (CVD) and hematological malignancies. In this Review, we will discuss seminal and recent studies that have advanced our understanding of mechanisms that drive selection for mutant HSPCs in the BM niche. Next, we will address recent studies evaluating potential relationships between the clonal dynamics of CHIP and hematopoietic development across the lifespan. Next, we will examine the roles of systemic factors that can influence hematopoietic stem cell (HSC) fitness, including inflammation, and exposures to cytotoxic agents in driving selection for CHIP clones. Furthermore, we will consider how - through their impact on the BM niche - lifestyle factors, including diet, exercise, and psychosocial stressors, might contribute to the process of somatic evolution in the BM that culminates in CHIP. Finally, we will review the role of old age as a major driver of selection in CHIP.

Indexed as

Clonal HematopoiesisHematopoietic Stem CellsStem Cell NicheAnimalsBone MarrowHumansMutation

Identifiers

PMID39087468
PMCPMC11290965

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.