Evidence mapPaperPMID 39093510Full record

ArticleMolecular biology reports2024

Swimming in cold water increases the browning process by diminishing the Myostatin pathway.

Niloofar Rahmani, Pezhman Motamedi, Sadegh Amani-Shalamzari, Kurt A Escobar, Katsuhiko Suzuki

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Article in Molecular biology reports, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

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2citing papers in PubMed
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2 citing papers in PubMed.

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5 · Who and what money

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5 authors.

Niloofar RahmaniDepartment of Exercise Physiology, Faculty of Physical Education and Sports Science, Kharazmi University, Tehran, Iran.
Pezhman MotamediDepartment of Exercise Physiology, Faculty of Physical Education and Sports Science, Kharazmi University, Tehran, Iran. pezhman.motamedi@khu.ac.ir.
Sadegh Amani-ShalamzariDepartment of Exercise Physiology, Faculty of Physical Education and Sports Science, Kharazmi University, Tehran, Iran.
Kurt A EscobarPhysiology of Exercise & Sport Lab, Department of Kinesiology, California State University, Long Beach, USA.
Katsuhiko SuzukiFaculty of Sport Sciences, Waseda University, Mikajima, Tokorozawa, Japan. katsu.suzu@waseda.jp.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundBrown adipose tissue (BAT) is a thermogenic tissue that uncouples oxidative phosphorylation from ATP synthesis and increases energy expenditure via non-shivering thermogenesis in mammals. Cold exposure and exercise have been shown to increase BAT and browning of white adipose tissue (WAT) in mice. This study aimed to determine whether there is an additive effect of exercise during cold exposure on markers related to browning of adipose tissue. in Wistar rats.

methodsTwenty-four male Wistar rats were randomly divided into three groups: Control (C, 25˚C), Swimming in Neutral (SN, 30˚C) water, and Swimming in Cold (SC, 15˚C) water. Swimming included intervals of 2-3 min, 1 min rest, until exhausted, three days a week for six weeks, with a training load of 3-6% body weight. After the experimental protocol, interscapular BAT and inguinal subcutaneous white adipose tissue (WAT) were excised, weighed, and processed for beiging marker gene expression.

resultsSN and SC resulted in lower body weight gain, associated with reduced WAT and BAT volume and increased BAT number with greater effects observed in SC. Myostatin protein expression was lower in BAT, WAT, soleus muscle, and serum NC and SC compared to the C group. Expression of the interferon regulatory factor-4 (IRF4) gene in both BAT and WAT tissues was significantly greater in the SC than in the C. Expression of the PGC-1α in BAT was significantly increased in the SC compared to C and increased in WAT in NC and SC. Expression of the UCP1 in BAT and WAT increased in the SC group compared to other groups.

conclusionThe findings demonstrate that six weeks of swimming training in cold water promotes additive effects of the expression of genes and proteins involved in the browning process of adipose tissue in Wistar rats. Myostatin inhibition may possess a regulator effect on the PGC-1α - UCP1 pathway that mediates adipose tissue browning.

Indexed as

Adipose Tissue, BrownAdipose Tissue, WhiteCold TemperatureMyostatinPhysical Conditioning, AnimalRats, WistarSwimmingThermogenesisAnimalsBody WeightEnergy MetabolismMalePeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaRatsSignal TransductionWaterMstn protein, ratMyostatinPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaWaterBrowningCold stressExercise trainingWhite fat

Identifiers

PMID39093510
PMCPMC11297105

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.