Evidence map›Paper›PMID 39097923›Full record

ArticleCNS neuroscience & therapeutics2024

Honokiol relieves hippocampal neuronal damage in Alzheimer's disease by activating the SIRT3-mediated mitochondrial autophagy.

Haitao Li, Jinmei Sun, Yili Wu, Yishu Yang, Wei Zhang, Yuanruhua Tian

Abstract read
In one paragraph

Article in CNS neuroscience & therapeutics, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed.

  1. Review
  2. Article
  3. cGAS-STING signaling in pollution-induced toxicity: mechanism and relieving medication.Apoptosis : an international journal on programmed cell death · 2026
    Review
  4. Review
  5. Review
  6. Review
  7. Review
  8. Article
  9. Article
  10. Irisin Regulates Microglia M1/M2 Polarization and Promotes Autophagy Through the Sirt3 Pathway to Alleviate POCD.Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology · 2025
    Article
  11. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Haitao LiDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China.ORCID 0000-0002-4416-7430
Jinmei SunDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China.
Yili WuZhejiang Provincial Clinical Research Center for Mental Disorders, School of Mental Health and The Affiliated Wenzhou Kangning Hospital, Institute of Aging, Key Laboratory of Alzheimer's Disease of Zhejiang Province, Wenzhou Medical University, Oujiang Laboratory (Zhejiang Lab for Regenerative Medicine, Vision and Brain Health), Wenzhou, China.
Yishu YangDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China.
Wei ZhangDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China.
Yuanruhua TianDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China.

Funding

Funding from Key Laboratory of Alzheimer's Disease of Zhejiang Province, Institute of Aging, Wenzhou Medical University ZJAD-2021003Hospital/Ten Majors/Special FinancialTen major professional constructions of Beijing Medical Management Center Q19051-07
6 · The paper itself

Abstract

backgroundThis work elucidated the effect of honokiol (HKL) on hippocampal neuronal mitochondrial function in Alzheimer's disease (AD).

methodsAPP/PS1 mice were used as AD mice models and exposed to HKL and 3-TYP. Morris water maze experiment was performed to appraise cognitive performance of mice. Hippocampal Aβ+ plaque deposition and neuronal survival was evaluated by immunohistochemistry and Nissl staining. Hippocampal neurons were dissociated from C57BL/6 mouse embryos. Hippocampal neuronal AD model was constructed by Aβ oligomers induction and treated with HKL, CsA and 3-TYP. Neuronal viability and apoptosis were detected by cell counting kit-8 assay and TUNEL staining. mRFP-eGFP-LC3 assay, MitoSOX Red, dichlorodihydrofluorescein diacetate, and JC-1 staining were performed to monitor neuronal autophagosomes, mitochondrial reactive oxygen species (ROS), neuronal ROS, and mitochondrial membrane potential. Autophagy-related proteins were detected by Western blot.

resultsIn AD mice, HKL improved cognitive function, relieved hippocampal Aβ

conclusionsHKL activates SIRT3-mediated mitochondrial autophagy to mitigate hippocampal neuronal damage in AD. HKL may be effective in treating AD.

Indexed as

Alzheimer DiseaseAmyloid beta-PeptidesAutophagyBiphenyl CompoundsHippocampusLignansMice, Inbred C57BLMice, TransgenicMitochondriaNeuronsSirtuin 3Allyl CompoundsAnimalsDisease Models, AnimalMaleMiceAllyl CompoundsAmyloid beta-PeptidesBiphenyl CompoundshonokiolLignansNeuroprotective AgentsPeptide FragmentsPhenolsReactive Oxygen SpeciesSirt3 protein, mouseSirtuin 3Alzheimer's diseasehonokiolmitochondrial autophagyneuronal damageSIRT3

Identifiers

PMID39097923
PMCPMC11298204

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.