Evidence map›Paper›PMID 39114641›Full record

ReviewFrontiers in molecular neuroscience2024

Wnt signaling pathway in spinal cord injury: from mechanisms to potential applications.

Kai Li, Zanzhi Chen, Xuejing Chang, Ruiyang Xue, Huaibo Wang, Weitao Guo

Abstract readReview
In one paragraph

Review in Frontiers in molecular neuroscience, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

  1. Review
  2. Article
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  6. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Kai LiDepartment of Spine Surgery, The Second Hospital Affiliated to Guangdong Medical University, Zhanjiang, China.
Zanzhi ChenDepartment of Spine Surgery, The Second Hospital Affiliated to Guangdong Medical University, Zhanjiang, China.
Xuejing ChangDepartment of Spine Surgery, The Second Hospital Affiliated to Guangdong Medical University, Zhanjiang, China.
Ruiyang XueDepartment of Spine Surgery, The Second Hospital Affiliated to Guangdong Medical University, Zhanjiang, China.
Huaibo WangDepartment of Spine Surgery, The Second Hospital Affiliated to Guangdong Medical University, Zhanjiang, China.
Weitao GuoDepartment of Spine Surgery, The Second Hospital Affiliated to Guangdong Medical University, Zhanjiang, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Spinal cord injury (SCI) denotes damage to both the structure and function of the spinal cord, primarily manifesting as sensory and motor deficits caused by disruptions in neural transmission pathways, potentially culminating in irreversible paralysis. Its pathophysiological processes are complex, with numerous molecules and signaling pathways intricately involved. Notably, the pronounced upregulation of the Wnt signaling pathway post-SCI holds promise for neural regeneration and repair. Activation of the Wnt pathway plays a crucial role in neuronal differentiation, axonal regeneration, local neuroinflammatory responses, and cell apoptosis, highlighting its potential as a therapeutic target for treating SCI. However, excessive activation of the Wnt pathway can also lead to negative effects, highlighting the need for further investigation into its applicability and significance in SCI. This paper provides an overview of the latest research advancements in the Wnt signaling pathway in SCI, summarizing the recent progress in treatment strategies associated with the Wnt pathway and analyzing their advantages and disadvantages. Additionally, we offer insights into the clinical application of the Wnt signaling pathway in SCI, along with prospective avenues for future research direction.

Indexed as

clinical applicationnerve regenerationneuroinflammationspinal cord injuryWnt signaling pathway

Identifiers

PMID39114641
PMCPMC11303303

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.