Evidence mapPaperPMID 39135805Full record

ArticleFrontiers in pharmacology2024

Melatonin supplementation counteracts fiber loss in knee ligaments of diabetes-induced rats.

Olga Adamska, Artur Wnuk, Agnieszka Kamińska, Małgorzata Poniatowska, Bartosz Maciąg, Maciej Kamiński, Artur Stolarczyk, Maima Matin, Atanas G Atanasov, Marcin Łapiński and 1 more

Abstract read
In one paragraph

Article in Frontiers in pharmacology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Olga AdamskaDepartment of Ophthalmology, Collegium Medicum, Cardinal Stefan Wyszynski University, Warsaw, Poland.
Artur WnukHospital in Ostrow Mazowiecka, Ostrów Mazowiecka, Poland.
Agnieszka KamińskaDepartment of Ophthalmology, Collegium Medicum, Cardinal Stefan Wyszynski University, Warsaw, Poland.
Małgorzata PoniatowskaDepartment of Nuclear Medicine, Pomeranian Medical University in Szczecin, Szczecin, Poland.
Bartosz MaciągOrthopedic and Rehabilitation Department, Medical University of Warsaw, Warsaw, Poland.
Maciej KamińskiMedical Faculty, Lazarski University, Warsaw, Poland.
Artur StolarczykOrthopedic and Rehabilitation Department, Medical University of Warsaw, Warsaw, Poland.
Maima MatinInstitute of Genetics and Animal Biotechnology of the Polish Academy of Sciences, Magdalenka, Poland.
Atanas G AtanasovInstitute of Genetics and Animal Biotechnology of the Polish Academy of Sciences, Magdalenka, Poland.
Marcin ŁapińskiOrthopedic and Rehabilitation Department, Medical University of Warsaw, Warsaw, Poland.
Artur JóźwikInstitute of Genetics and Animal Biotechnology of the Polish Academy of Sciences, Magdalenka, Poland.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Diabetes mellitus (DM) is a prevalent metabolic disease. The clinical impact of sustained hyperglycemia on ligament healing has not been well characterized. Diabetes is a known cause of macro-, microvascular, and diabetic ulcer healing difficulties among tissues. Therefore, we aimed to investigate the healing potential occurring in injured and healthy ligaments among diabetic and healthy individuals using a rat model. We hypothesize that DM may contribute to altering the knee medial collateral ligament (MCL), thus its morphology, biochemical fitness, and functionality. The study cohort consisted of 40 rats. The animals were randomized into four equal groups. Groups I and II (20 rats) received saline subcutaneously and served as controls. Groups III and IV (20 rats) were injected with a single dose of streptozotocin (STZ). All animals underwent surgery to cut the left tibial collateral ligament in the hind limb and suture it. The access site was sutured to create inflammation and study the regenerative capacities of animals with normal carbohydrate metabolism and pharmacologically induced diabetes. Each animal then underwent sham surgery to access and suture the right tibial collateral ligament in the hind limb without ligament intervention. After the animals had undergone surgeries, groups II and IV were given melatonin supplementation for 4 weeks. Rats with DM presented with more fibrosis and calcification of the MCL and decreased healing potential. Treatment with melatonin in diabetic rats mitigated alterations and improved the antioxidant status of ligaments from the diabetic group.

Indexed as

diabetes ligamentsdiabetes mellitusligaments alterationligaments alteration in diabetes mellitusorthopedic complications

Identifiers

PMID39135805
PMCPMC11317382

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.