Evidence mapPaperPMID 39148278Full record

ReviewAging cell2024

The connection between aging, cellular senescence and gut microbiome alterations: A comprehensive review.

Dong-Hyun Jang, Ji-Won Shin, Eunha Shim, Naoko Ohtani, Ok Hee Jeon

Abstract readReview
In one paragraph

Review in Aging cell, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 49 papers.

0numbers the graph read from it
0cells of the map it votes in
49citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

49 citing papers in PubMed.

  1. Review
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  5. Translational Geroscience Strategies for Delaying Multimorbidity.ACS pharmacology & translational science · 2026
    Review
  6. Article
  7. Review
  8. Article
  9. Review
  10. Review
  11. Review
  12. Review
  13. The gut as a central hub for multi-organ crosstalk in aging.Cellular and molecular life sciences : CMLS · 2026
    Review
  14. Article
  15. Article
  16. Review
  17. Review
  18. Review
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Dong-Hyun JangDepartment of Biomedical Sciences, Korea University College of Medicine, Seoul, Republic of Korea.ORCID 0009-0009-8018-6637
Ji-Won ShinDepartment of Biomedical Sciences, Korea University College of Medicine, Seoul, Republic of Korea.ORCID 0000-0002-4114-9317
Eunha ShimDepartment of Biomedical Sciences, Korea University College of Medicine, Seoul, Republic of Korea.ORCID 0009-0001-6119-123X
Naoko OhtaniDepartment of Pathophysiology, Osaka Metropolitan University Graduate School of Medicine, Osaka, Japan.ORCID 0000-0001-8934-0797
Ok Hee JeonDepartment of Biomedical Sciences, Korea University College of Medicine, Seoul, Republic of Korea.ORCID 0000-0002-3725-7526

Funding

Korean government (MSIT) RS-2023-00220894National Research Foundation of Korea (NRF) 2020R1C1C1009921National Research Foundation of Korea (NRF) RS2024-00340798
6 · The paper itself

Abstract

The intricate interplay between cellular senescence and alterations in the gut microbiome emerges as a pivotal axis in the aging process, increasingly recognized for its contribution to systemic inflammation, physiological decline, and predisposition to age-associated diseases. Cellular senescence, characterized by a cessation of cell division in response to various stressors, induces morphological and functional changes within tissues. The complexity and heterogeneity of senescent cells, alongside the secretion of senescence-associated secretory phenotype, exacerbate the aging process through pro-inflammatory pathways and influence the microenvironment and immune system. Concurrently, aging-associated changes in gut microbiome diversity and composition contribute to dysbiosis, further exacerbating systemic inflammation and undermining the integrity of various bodily functions. This review encapsulates the burgeoning research on the reciprocal relationship between cellular senescence and gut dysbiosis, highlighting their collective impact on age-related musculoskeletal diseases, including osteoporosis, sarcopenia, and osteoarthritis. It also explores the potential of modulating the gut microbiome and targeting cellular senescence as innovative strategies for healthy aging and mitigating the progression of aging-related conditions. By exploring targeted interventions, including the development of senotherapeutic drugs and probiotic therapies, this review aims to shed light on novel therapeutic avenues. These strategies leverage the connection between cellular senescence and gut microbiome alterations to advance aging research and development of interventions aimed at extending health span and improving the quality of life in the older population.

Indexed as

AgingCellular SenescenceGastrointestinal MicrobiomeAnimalsDysbiosisHumansagingcellular senescencegut microbiomemusculoskeletal diseasessenotherapeutics

Identifiers

PMID39148278
PMCPMC11464129

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.