Evidence mapPaperPMID 39150512Full record

ArticleActa diabetologica2025

Pancreatic beta cell function and insulin resistance profiles in first-degree relatives of patients with prediabetes and type 2 diabetes.

Anaid Herrerías-García, Emmanuel Jacobo-Tovar, Claudia Mariana Hernández-Robles, Rodolfo Guardado-Mendoza

Abstract read
PubMed Publisher
In one paragraph

Article in Acta diabetologica, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Trial
  2. Article
  3. Review
  4. Article
  5. Article
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Anaid Herrerías-GarcíaMetabolic Research Laboratory, Department of Medicine and Nutrition, University of Guanajuato, Blvd. Milenio 1001, Predio San Carlos, 37670, León, Guanajuato, Mexico.
Emmanuel Jacobo-TovarMetabolic Research Laboratory, Department of Medicine and Nutrition, University of Guanajuato, Blvd. Milenio 1001, Predio San Carlos, 37670, León, Guanajuato, Mexico.
Claudia Mariana Hernández-RoblesMetabolic Research Laboratory, Department of Medicine and Nutrition, University of Guanajuato, Blvd. Milenio 1001, Predio San Carlos, 37670, León, Guanajuato, Mexico.
Rodolfo Guardado-MendozaMetabolic Research Laboratory, Department of Medicine and Nutrition, University of Guanajuato, Blvd. Milenio 1001, Predio San Carlos, 37670, León, Guanajuato, Mexico. rguardado@ugto.mx.

Funding

University of Guanajuato 010/2014 and 018/2015
6 · The paper itself

Abstract

aimsTo evaluate insulin secretion and insulin resistance profiles in individuals with family history of prediabetes and type 2 diabetes.

methodsThis was a cross-sectional study to evaluate clinical and metabolic profiles between individuals with type 2 diabetes, prediabetes and their relatives. There were 911 subjects divided into five groups: (i) normoglycemic (NG), (ii) type 2 diabetes, (iii) prediabetes, (iv) first-degree relatives of patients with type 2 diabetes (famT2D), and (v) first-degree relatives of patients with prediabetes (famPD); anthropometrical, biochemical and nutritional evaluation, as well as insulin resistance and pancreatic beta cell function measurement was performed by oral glucose tolerance to compare between groups.

resultsThe most prevalent type 2 diabetes risk factors were dyslipidemia (81%), family history of type 2 diabetes (76%), central obesity (73%), male sex (63%), and sedentary lifestyle (60%), and most of them were progressively associated to prediabetes and type 2 diabetes groups. Insulin sensitivity was lower in famT2D groups in comparison to NG group (p < 0.0001). FamPD and famT2D had a 10% lower pancreatic beta cell function (DI) than the NG group (NG group 2.78 ± 1.0, famPD 2.5 ± 0.85, famT2D 2.4 ± 0.75, p˂0.001).

conclusionsFamPD and famT2D patients had lower pancreatic beta cell function than NG patients, highlighting that defects in insulin secretion and insulin sensitivity appear long time before the development of hyperglycemia in patients genetically predisposed.

Indexed as

Diabetes Mellitus, Type 2Insulin ResistanceInsulin-Secreting CellsPrediabetic StateAdultAgedCross-Sectional StudiesFamilyFemaleGlucose Tolerance TestHumansInsulinInsulin SecretionMaleMiddle AgedRisk FactorsInsulinGenetic predispositionInsulin sensitivityPancreatic beta cell dysfunctionPrediabetesType 2 diabetes

Identifiers

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.