Evidence map›Paper›PMID 39185405›Full record

ReviewFrontiers in immunology2024

Inflammation mechanism and research progress of COPD.

Jiao Xu, Qingyue Zeng, Shuangqing Li, Qiaoli Su, Hong Fan

Abstract readReview
In one paragraph

Review in Frontiers in immunology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 147 papers, 9 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
147citing papers in PubMed, 9 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

147 citing papers in PubMed, 9 syntheses or guidelines pooled it.

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  4. International journal of chronic obstructive pulmonary disease · 2026
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  16. Development of Wedelia (Current issues in molecular biology · 2026
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87 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Jiao Xu *General Practice Medical Center, West China Hospital, Sichuan University, Chengdu, China.
Qingyue Zeng *General Practice Medical Center, West China Hospital, Sichuan University, Chengdu, China.
Shuangqing LiGeneral Practice Medical Center, West China Hospital, Sichuan University, Chengdu, China.
Qiaoli SuGeneral Practice Medical Center, West China Hospital, Sichuan University, Chengdu, China.
Hong FanDepartment of Respiratory and Critical Care Medicine, West China Hospital, Sichuan University, Chengdu, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Chronic obstructive pulmonary disease (COPD) is a common respiratory disease characterized by irreversible progressive airflow limitation, often manifested by persistent cough, sputum production and other respiratory symptoms that pose a serious threat to human health and affect the quality of life of patients. The disease is associated with chronic inflammation, which is associated with the onset and progression of COPD, but anti-inflammatory therapy is not first-line treatment. Inflammation has multiple manifestations and phenotypes, and this heterogeneity reveals different patterns of inflammation, making treatment difficult. This paper aims to explore the direction of more effective anti-inflammatory treatment by analyzing the nature of inflammation and the molecular mechanism of disease occurrence and development in COPD patients, and to provide new ideas for the treatment of COPD patients.

Indexed as

InflammationPulmonary Disease, Chronic ObstructiveAnimalsAnti-Inflammatory AgentsHumansAnti-Inflammatory Agentschronic inflammationchronic obstructive pulmonary diseaseinflammatory cellsoxidative stresssystemic inflammation

Identifiers

PMID39185405
PMCPMC11341368

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.