Evidence mapPaperPMID 39192284Full record

ArticleClinical epigenetics2024

Mendelian randomization analysis reveals the combined effects of epigenetics and telomere biology in hematologic cancers.

Xin Zhuang, Peng Chen, Rong Yang, Xiaoying Man, Ruochen Wang, Yifen Shi

Abstract read
In one paragraph

Article in Clinical epigenetics, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Review
  2. [Causal association between gut microbiota and food allergy: a Mendelian randomization analysis].Zhongguo dang dai er ke za zhi = Chinese journal of contemporary pediatrics · 2025
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Xin ZhuangDepartment of Hematology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, China.
Peng ChenDepartment of Hematology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, China.
Rong YangDepartment of Hematology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, China.
Xiaoying ManDepartment of Hematology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, China.
Ruochen WangDepartment of Hematology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, China.
Yifen ShiDepartment of Hematology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, China. shiyifen1999@wzhospital.cn.

Funding

Natural Science Foundation of Zhejiang Province No. LQ19H080002the Public Welfare Science and Technology Project of Wenzhou Y20240077
6 · The paper itself

Abstract

backgroundTelomere shortening and epigenetic modifications are key factors in aging and hematologic diseases. This study investigates the relationship of telomere length and epigenetic age acceleration (EAA) with hematologic cancers, blood cells, and biochemical markers through the epigenetic clocks.

methodsThis study primarily utilizes genome-wide association studies of populations of European descent as instrumental variables, exploring the causal relationships between exposures and outcomes through a bidirectional two-sample Mendelian randomization (MR) approach. MR techniques include inverse variance weighted (IVW), MR Egger, and weighted median modes. Heterogeneity and pleiotropy in MR are assessed using Cochran's Q test and the MR Egger intercept, with the robustness of the conclusions further validated by multivariable MR (MVMR).

resultsOur research shows that longer telomere lengths significantly increase the risk of multiple myeloma, leukemia, and lymphoma (OR > 1, P < 0.05) and establish a causal relationship between telomere length and red blood cell indices such as RBC (OR = 1.121, P

conclusionThe study clarifies the relationships between telomere length, EAA, and hematological malignancies, further emphasizing the prognostic significance of telomere length and EAA. This deepens our understanding of the pathogenesis of hematological diseases, which can inform risk assessment and therapeutic strategies.

Indexed as

Epigenesis, GeneticGenome-Wide Association StudyHematologic NeoplasmsMendelian Randomization AnalysisTelomereDNA MethylationFemaleHumansMaleTelomere HomeostasisTelomere ShorteningEpigenetic age acceleration (EAA)Epigenetic clocksGenome-Wide Association Study (GWAS)Hematologic malignanciesMendelian randomization (MR)Telomere length

Identifiers

PMID39192284
PMCPMC11351094

What Socratic holds

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LicenceCC BY-NC-ND
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.