Evidence mapPaperPMID 39199338Full record

ArticleBiomolecules2024

The Role of Melatonin in the Inflammatory Process in Patients with Hyperglycemia and Leishmania Infection.

Thalissa Mariana de Moraes Martins, Felipe Rubin Ferrari, Adriele Ataides de Queiroz, Letícia Damas Leão Dalcin, Danielle Cristina Honorio França, Adenilda Cristina Honório-França, Eduardo Luzía França, Danny Laura Gomes Fagundes-Triches

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Article in Biomolecules, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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4 · The record

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5 · Who and what money

Authors and funding

8 authors.

Thalissa Mariana de Moraes MartinsInstitute of Biological and Health Science, Federal University of Mato Grosso, Barra do Garças 78605-091, MT, Brazil.
Felipe Rubin FerrariInstitute of Biological and Health Science, Federal University of Mato Grosso, Barra do Garças 78605-091, MT, Brazil.
Adriele Ataides de QueirozInstitute of Biological and Health Science, Federal University of Mato Grosso, Barra do Garças 78605-091, MT, Brazil.ORCID 0000-0001-9185-7429
Letícia Damas Leão DalcinInstitute of Biological and Health Science, Federal University of Mato Grosso, Barra do Garças 78605-091, MT, Brazil.
Danielle Cristina Honorio FrançaInstitute of Biological and Health Science, Federal University of Mato Grosso, Barra do Garças 78605-091, MT, Brazil.ORCID 0000-0002-4921-4064
Adenilda Cristina Honório-FrançaInstitute of Biological and Health Science, Federal University of Mato Grosso, Barra do Garças 78605-091, MT, Brazil.
Eduardo Luzía FrançaInstitute of Biological and Health Science, Federal University of Mato Grosso, Barra do Garças 78605-091, MT, Brazil.
Danny Laura Gomes Fagundes-TrichesInstitute of Biological and Health Science, Federal University of Mato Grosso, Barra do Garças 78605-091, MT, Brazil.ORCID 0000-0002-9971-1288

Funding

Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq) No 312511/2023-0; No 312841/2023-0Coordenação de Aperfeiçoamento de Pessoal de Nível Superior-Brazil (CAPES) code 001
6 · The paper itself

Abstract

Type 2 diabetes mellitus is a metabolic disorder that causes chronic high blood sugar levels, and diabetic patients are more susceptible to infections. American cutaneous leishmaniasis is an infectious disease caused by a parasite that affects the skin and mucous membranes, leading to one or multiple ulcerative lesions. Chronic inflammation and functional changes in various organs and systems, including the immune system, are the primary causes of both diseases. Melatonin, an essential immunomodulatory, antioxidant, and neuroprotective agent, can benefit many immunological processes and infectious diseases, including leishmaniasis. Although, limited reports are available on diabetic patients with leishmaniasis. The literature suggests that melatonin may play a promising role in inflammatory disorders. This study was designed to assess melatonin levels and inflammatory mediators in diabetic patients affected by leishmaniasis. Blood samples from 25 individuals were analyzed and divided into four groups: a control group (without any diseases), a Leishmania-positive group, patients with type 2 diabetes mellitus, and patients with a combination of both diseases. This study measured the serum levels of melatonin through ELISA, while IL-4 and TNF-α were measured using flow cytometry, and C-reactive protein was measured through turbidimetry. This study found that patients with leishmaniasis significantly increased TNF-α and decreased melatonin levels. However, the group of diabetic patients with leishmaniasis showed higher melatonin levels than the control group. These observations suggest that TNF-α may influence melatonin production in patients with American cutaneous leishmaniasis, potentially contributing to the inflammatory characteristics of both diseases.

Indexed as

Diabetes Mellitus, Type 2HyperglycemiaInflammationMelatoninTumor Necrosis Factor-alphaAdultAgedC-Reactive ProteinFemaleHumansInterleukin-4LeishmaniasisLeishmaniasis, CutaneousMaleMiddle AgedC-Reactive ProteinIL4 protein, humanInterleukin-4MelatoninTumor Necrosis Factor-alphacytokinesdiabetesinflammationleishmaniasismelatonin

Identifiers

PMID39199338
PMCPMC11352828

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.